The Playground

  • This means that roughly half of Switzerland’s Covid deaths over the last 4 weeks have occurred among a relatively small number of unvaccinated people.

    And now. What does this tell? Only unvaccinated die? OR as you said 8x more unvaccinated die?? You statement just works in case of fake subgrouping.


    And guess > 50% of these vaccinated are protected by an infection. So also then its just 4x...

  • And now. What does this tell? Only unvaccinated die? OR as you said 8x more unvaccinated die?? You statement just works in case of fake subgrouping.

    Almost ALL deaths (not a fake subgroup) are people over 60…

    It’s not about vaxxed and vaxxed infected that might be somehow protected. The report and the numbers are about UNVAXXED, and those dye at a much (8x) higher rate if they get infected. It is all about the risk of dying and being vaccinated or not. But those numbers prove efficiency of vaccines and therefore don’t fit in your philosophy of course.

  • ‘There is no Control of the Omicron Wave’ as Heavily Pfizer-Boosted Nation Shatters Infection Record


    ‘There is no Control of the Omicron Wave’ as Heavily Pfizer-Boosted Nation Shatters Infection Record
    The omicron variant of SARS-CoV-2 rages now through the eastern Mediterranean nation of Israel as the reported number of cases hit a COVID-19 pandemic
    trialsitenews.com


    The omicron variant of SARS-CoV-2 rages now through the eastern Mediterranean nation of Israel as the reported number of cases hit a COVID-19 pandemic record of 17,362 new infections on Monday, January 5th. As TrialSite just reported, a fourth booster program commences, targeting higher risk demographics such as the elderly and immunocompromised. This sweeping contagion, while seemingly milder in symptoms, demonstrates the limitations of the mass vaccination to eradicate SARS-CoV-2 strategy in this heavily vaccinated nation driven by a confluence of elite medical establishment officials and executives in regulatory agencies, national and international public health authorities, elite academic medical centers, and industry not to mention financiers and investors monetizing the pandemic and influential not-for-profits. TrialSite shared with community members that by October and November of last year, based on Israel Health Ministry data that the unvaccinated still faced a higher risk for severe infection, but we also reported on striking numbers of breakthrough infections leading to hospitalization. One Sheba Medical Center study opens up a Moderna arm—are there some underlying seeds of doubt concerning Pfizer-BioNTech’s BNT162b2?


    The good news is that despite historically high infections, the death rate is very low, at least thus far, based on data from the COVID-19 Data Repository by the Center for Systems Science and Engineering (CSSE) at Johns Hopkins University. TrialSite cannot be certain what’s behind the lower death rate but more than likely, a confluence of factors from vaccination and growing natural immunity to the milder mutant could be an explanation. So, in that way, mass vaccine proponents will claim that the products made the pandemic milder but a fundamental underlying assumption for what is now hundreds of billions worldwide spent on vaccine-supporting programs was very much centered on pathogen eradication—something the vaccine products are failing to address. TrialSite reminds all that, worldwide, nearly 5.5 million Covid deaths are recorded. Many critical care physicians have argued all along that off-label use of a handful of repurposed drugs could have saved many lives.


    Out of Control

    Sharon Alroy-Preis, the nation’s top public health official, told Channel 13, “There is no control of the Omicron wave.” The health minister continued, “Probably no one is protected from infection,” reports the president of Shaare Zedek Medical Center in Jerusalem.


    Goal: Keep Economy Open

    Avoidance of economic pain and suffering becomes a top goal of governments across the West as the realities of the situation settle in—meaning the transition to an endemic stage and learning to live with the SARS-CoV-2 pathogen. Prime Minister Naftali Bennett declared at a recent press conference, “It’s a different ball game altogether.”



    Let the Boosts Begin

    Regardless of vaccine limitation, the fourth boost program accelerates full throttle. Israel21c reports thousands started last week getting their fourth booster shot of the Pfizer-BioNTech mRNA-based vaccine since the government authorized use for healthcare workers, the elderly, and immunocompromised. The rationale for the decision? Protect the vulnerable from the imminent omicron wave. The decision was made prior to an important Sheba Medical Center study investigating the impact of a fourth dose on 150 employees at the academic medical center.


    Israeli media reports that all those subjects that received three Pfizer-BioNTech doses five or more months ago now don’t have sufficient antibodies to respond to SARS-CoV-2.


    Concern with Pfizer-BioNTech Vaccine?

    Interestingly, in this Sheba Medical Center study, led by the director of the Infectious Disease Epidemiology Unit, the study now expands, and a hospital spokesperson told Israel21c that they will embrace the Moderna mRNA-based vaccine on a separate group of study subjects. They will test if mRNA-1273 has a superior impact to resist omicron in healthcare workers that have already received three Pfizer-BioNTech doses

  • It’s not about vaxxed and vaxxed infected that might be somehow protected. The report and the numbers are about UNVAXXED, and those dye at a much (8x) higher rate if they get infected

    Oh yeas 7 death age < 50 of 250.... great. Same number we have from flu....


    So you basically agree that the vaccines add nothing / do not help much....But our terror friends tell a different story...


    Not to mention treatment and the much higher number of gene therapy death/cripples age < 50...

  • Japan has basically been locked down..no foreigners allowed

    except in Okinawa....was it the mice?

    U.S. servicemen. They come in by military flights. They are allowed anytime. The mayors of Hiroshima and Iwakuni are hopping mad at the U.S. for not controlling the pandemic at Iwakuni airbase, and for not telling the Japanese government exactly how many soldiers are infected and their case status.


    ~240 cases in Iwakuni:


    80 new COVID infections confirmed at US Marines' Iwakuni base in Japan - The Mainichi
    YAMAGUCHI -- Eighty coronavirus infections have been newly confirmed among personnel at U.S. Marine Corps Air Station Iwakuni, the Yamaguchi Prefectur
    mainichi.jp


    Latest coronavirus wave grows higher at US military bases across Japan
    Marine Corps installations in Japan reported the bulk of the 242 new cases reported by the U.S. military in the country as the winter coronavirus surge…
    www.stripes.com

  • The magician writes:

    Actually the video does very poor, which is due to the effective measures that YT has to prevent fraud. So his paid minions only can manipulate the numbers on Researchgate.

  • IMO a seriously made documentary talking about situation in Guatemala, Isreal, Kenia compared especially to Europe in a none main stream view.


    Corona – auf der Suche nach der Wahrheit, Teil 3: Die Themen
    Auch in Teil drei der Corona-Doku-Reihe begibt sich Prof. Dr. Dr. Haditsch auf eine Reise um die Welt. Das sind die Themen.
    www.servustv.com


    Unfortunatley only in German.

    Some parts are in English, pay attention to the Israeli doctor of the largest hospital in IL and situation with IVM, Dr. Francesco ZAMBON WHO whistleblower, Prof. Christian PERRONE and others....

  • Japan has basically been locked down..no foreigners allowed

    except in Okinawa....was it the mice?

    https://www.asahi.com/ajw/articles/14514604

    Evidence for a mouse origin of the SARS-CoV-2 Omicron variant


    Evidence for a mouse origin of the SARS-CoV-2 Omicron variant
    The rapid accumulation of mutations in the SARS-CoV-2 Omicron variant that enabled its outbreak raises questions as to whether its proximal origin occurred in…
    www.biorxiv.org


    ABSTRACT

    The rapid accumulation of mutations in the SARS-CoV-2 Omicron variant that enabled its outbreak raises questions as to whether its proximal origin occurred in humans or another mammalian host. Here, we identified 45 point mutations that Omicron acquired since divergence from the B.1.1 lineage. We found that the Omicron spike protein sequence was subjected to stronger positive selection than that of any reported SARS-CoV-2 variants known to evolve persistently in human hosts, suggesting the possibility of host-jumping. The molecular spectrum (i.e., the relative frequency of the twelve types of base substitutions) of mutations acquired by the progenitor of Omicron was significantly different from the spectrum for viruses that evolved in human patients, but was highly consistent with spectra associated with evolution in a mouse cellular environment. Furthermore, mutations in the Omicron spike protein significantly overlapped with SARS-CoV-2 mutations known to promote adaptation to mouse hosts, particularly through enhanced spike protein binding affinity for the mouse cell entry receptor. Collectively, our results suggest that the progenitor of Omicron jumped from humans to mice, rapidly accumulated mutations conducive to infecting that host, then jumped back into humans, indicating an inter-species evolutionary trajectory for the Omicron outbreak.


    DISCUSSION

    In this study, we used the molecular spectrum of mutations of the SARS-CoV-2 Omicron variant to trace its proximal host origins. We found that the molecular spectrum of pre-outbreak Omicron mutations was inconsistent with the rapid accumulation of mutations in humans, but rather suggested a trajectory in which the progenitor of Omicron experienced a reverse zoonotic event from humans to mice sometime during the pandemic (most likely in mid-2020) and accumulated mutations in a rodent host (most likely mouse) for more than one year before jumping back to humans in late-2021. While evolving in mice, the progenitor of Omicron adapted to the mouse host by acquiring amino acid mutations in the spike protein that increased its binding affinity with mouse ACE2. In addition, mutations associated with immune escape also accumulated, which may also be a contributing factor in its rapid spread.


    While we show a phylogenetically long branch leading to the MRCA of current Omicron variants (i.e., Branch O), it is worth noting that intermediate versions of Omicron were occasionally reported. For example, a SARS-CoV-2 variant (EPI_ISL_7136300) was collected by the Utah Public Health Laboratory on December 1st, 2021 which harbored 32 of the 45 pre-outbreak Omicron mutations. However, the 13 mutations absent in this variant clustered within residues 371–501 of the spike protein (Fig. S3). The absence of these spike protein mutations thus suggested that this variant was a product of recombination between an Omicron variant and another SARS-CoV-2 variant, rather than a direct progenitor of Omicron. Considering the large number of pre-outbreak Omicron mutations (45) combined with the sparsity of intermediate versions identified to date, this long branch leading to Omicron in our phylogenetic reconstruction remains valid.


    Although we primarily focused on point mutations because the molecular spectrum of these mutations can reflect the host cellular environment (Deng et al., 2021; Shan et al., 2021), we also used the information of deletions and insertions to infer the evolutionary trajectory of Omicron. For example, a B.1.1 variant (EPI_ISL_493480) shared the same deletion (Δ105–107 in non-structural protein 6) as the Omicron variants, which was used to infer that B.1.1 is a close relative of Omicron. In addition, spike Δ69–70 deletion is shared by Omicron and many non-Omicron variants isolated from patients (Meng et al., 2021), but is absent in the early samples of SARS-CoV-2 (Wu et al., 2020a), strongly suggesting that the progenitor of Omicron was jumped from humans to mice during the pandemic.


    In addition, we noted that Omicron harbored a nine nucleotide insertion (GAGCCAGAA, encoding the peptide EPE) after residue 214 in the spike protein. This insertion is identical to the sequence of TMEM245 in the human genome or that of ORF S in the human coronavirus hCoV-229E, which was used as evidence to support a human origin for Omicron (Venkatakrishnan et al., 2021). However, we provide a simpler explanation for this insertion, namely that it was derived from an RNA fragment of ORF N in the SARS-CoV-2 genome (Fig. S4). We believe that the insertion of an ORF N fragment is more likely because the RNA abundance of ORF N is much higher than that of mRNA encoded by the human genome (Wei et al., 2021). That is, in SARS-CoV-2-infected cells, a substantial proportion of RNAs are viral, and especially so for ORF N due to the nested nature of the coronavirus genome and subgenomes (Kim et al., 2020).


    It also warrants mention that all of the mouse-adapted SARS-CoV-2 variants were amplified/purified in Vero cells (a cell line originally isolated from the kidney of green monkey) at some stage of experimentation, which could impose an additional selection pressure to enhance the spike protein binding affinity towards primate ACE2 (Leist et al., 2020; Wu et al., 2020b; Huang et al., 2021; Montagutelli et al., 2021; Sun et al., 2021; Wong et al., 2021; Zhang et al., 2021). Consistent with this experimental process, the amino acid mutations acquired by Omicron and mouse-adapted viruses were not always identical, even if mutations occurred at the same residue. For example, Q493H and Q493K were also detected in the mouse-adapted SARS-CoV-2 at residue 493 of the spike protein, in addition to mutations observed in Omicron (Q493R). Different from the effects of Q493R, these two mutations increased the binding affinity toward both mouse and human ACE2 (Fig. S2C), indicating that SARS-CoV-2 could potentially evolve remarkably high diversity in its adaptation to ACE2 from various host species. Consistent with this possibility, numerous mutations were also identified in the spike protein of SARS-CoV-2 RNA fragment amplified from wastewater samples (Smyth et al., 2021).


    Humans represent the largest known reservoir of SARS-CoV-2. Our study suggests that SARS-CoV-2 could have spilled over from humans to wild animals, and that the variants which successfully infected animal hosts could then accumulate new mutations before jumping back into humans as a variant of concern. Given the ability of SARS-CoV-2 to jump across various species, it appears likely that global populations will face additional animal-derived variants until the pandemic is well under control. Viral surveillance and sequencing in wild animals will likely help to prevent future outbreaks of dangerous SARS-CoV-2 variants.

  • Corona – auf der Suche nach der Wahrheit, Teil 3: Die Themen
    Auch in Teil drei der Corona-Doku-Reihe begibt sich Prof. Dr. Dr. Haditsch auf eine Reise um die Welt. Das sind die Themen.
    www.servustv.com


    Fm1 in above documentary is also an exclusive interview of Dr. ZAMBON WHO whistleblower in English! A brave man! Starts at around 24:30


    One day, the truth cannot be hidden anymore and comes onto the table!

  • Mystery of long COVID-19 is solved! This scientist believes so


    `Mystery of long COVID-19 is solved! This scientist believes so , Science News | wionews.com


    Globally, about 294 million people have been infected with the coronavirus, with more than 5.8 million deaths.Since the first cases were discovered in central China in December 2019, infections have been reported in over 210 countries and territories.


    Now, a South African scientist believes she and her colleagues have discovered a crucial answer to the enigma of long COVID


    In an op-ed published Wednesday, Resia Pretorius, the head of the science department at Stellenbosch University in South Africa, wrote, "A recent investigation in my lab found that there is significant microclot formation in the blood of both acute COVID-19 and long COVID patients.



    She discovered a correlation between microclots and lengthy COVID, which she believes is a critical step toward better understanding the disease that affects 100 million people worldwid


    Resia Pretorius and her team discovered large quantities of inflammatory chemicals "locked" in the persistent microclots seen in extended COVID patients during the summe


    In June, the Department of Human and Health Services issued new guidelines that define some long-term COVID symptoms as an impairment under the Americans with Disabilities Act. r.e.ns" .

    According to the Pretorius's op-ed "Patients with long Covid complain of numerous symptoms, the main ones being recurring fatigue and brain fog, muscle weakness, being out of breath and having low oxygen levels, sleep difficulties and anxiety or depression. Some patients are so sick that they cannot work or even walk a few steps. There is possibly also an elevated risk of stroke and heart attacks. One of the biggest sources of concern is that even mild and sometimes asymptomatic initial Covid-19 infection may lead to debilitating, long-term disability."


    According to a study published in November by researchers at the University of Michigan, as many as 100 million people worldwide have or have had lengthy COVID.


    According to the Mayo Clinic, a patient is diagnosed with protracted COVID when the consequences of a COVID-19 infection last for longer than four weeks.


    While elderly persons and those with major medical issues are more prone to have protracted COVID, many young and healthy people have reported feeling ill for weeks or even months after being diagnosed with COVID-19.

  • Flu season has just started in Japan and Covid cases are again on the rise. I told you so!


    Japan's Okinawa sees doubling of COVID-19 cases, considers emergency steps


    Japan's Okinawa sees doubling of COVID-19 cases, considers emergency steps
    The southern Japanese island chain of Okinawa emerged on Wednesday as the epicentre of a new coronavirus surge, with cases more than doubling from the previous…
    www.reuters.com


    TOKYO, Jan 5 (Reuters) - The southern Japanese island chain of Okinawa emerged on Wednesday as the epicentre of a new coronavirus surge, with cases more than doubling from the previous day and officials were considering imposing emergency curbs.

  • Omicron infections seen in 46% of cases in Japan


    https://the-japan-news.com/news/article/0008160174


    Nearly half of the novel coronavirus infections reported nationwide in a recent seven-day period were believed to be omicron variant cases, the Health, Labor and Welfare Ministry said Thursday.


    Among about 2,000 infected people nationwide who underwent PCR testing for the seven days through Jan. 2, the highly contagious variant was suspected in 46% of cases, the ministry said at a meeting of an advisory panel analyzing the infection situation.


    The advisory body called for caution, saying the medical system could be placed under strain if the number of people infected with omicron surges, because such cases have now been confirmed in about 80% of all prefectures.


    The ministry’s analysis indicated that omicron is replacing other forms of the virus mainly in western Japan.


    By prefecture, Okinawa, which is facing another surge of new cases, saw the variant accounting for 73% of cases over the period. It was followed by Hiroshima with 75%, Yamaguchi with 71%, Kyoto with 64% and Osaka with 60%.


    In addition, prefectures in urban areas had higher percentages of cases with the variant, such as Aichi with 72% and Chiba with 51%, according to the ministry.


    At the meeting, the severity of COVID-19 symptoms in 675 people in Okinawa Prefecture, most of whom were believed to be infected with omicron, was reported. Of them, 92.3% have had no or mild symptoms, while 7.7% were moderately ill, and no one fell into serious condition.


    An analysis of 50 people who were confirmed to be infected with omicron found that 72% developed fever of 37.5 C or higher, while 58% had coughing, 50% suffered fatigue and 44% complained sore throat. There were a smaller number of cases that reported difficulties in breathing or loss of taste.

  • Oh yeas 7 death age < 50 of 250.... great. Same number we have from flu....


    So you basically agree that the vaccines add nothing / do not help much....But our terror friends tell a different story...


    Not to mention treatment and the much higher number of gene therapy death/cripples age < 50...

    Who was talking about deaths <50 ?.. can’t you read what has been posted? Twisting facts and comparing something with the flu death rate <50 was not the topic at all …

    If you can (and want to?) READ what is in your official Swiss Covid reports, you can find what everyone else can…


    Here once again for you:


    „Over the 4 weeks to 14 December 2021, 525 Covid-19 deaths were recorded in Switzerland. These were split roughly equally between the vaccinated and unvaccinated. However, to make sense of these numbers it is essential to look at the number of people in each group. Once this is factored in the life-saving power of vaccines is clear.

    Nearly all of those dying were 60 or over2. Among this age group, 88% were fully vaccinated, 1% partially vaccinated and 11% unvaccinated. This means that roughly half of Switzerland’s Covid deaths over the last 4 weeks have occurred among a relatively small number of unvaccinated people. In fact, the unvaccinated in this age group (60+) have been dying at a rate 7.53 times higher than the vaccinated, essentially because the vaccinated now outnumber the unvaccinated by 7.5 to 1.“

  • She discovered a correlation between microclots and lengthy COVID, which she believes is a critical step toward better understanding the disease that affects 100 million people worldwid

    We already linked the cure here:: They are a bit late.....https://www.researchgate.net/publication/357428572


    Omicron causes no damage at least in the hamster model that have been highly sensitive to delta...


    What Japan's latest research tells us about omicron variant risks - The Mainichi
    TOKYO -- It appears highly unlikely that the coronavirus's omicron variant spreading across the country will make people as sick as the delta variant
    mainichi.jp


    Here in CH despite 3x cases hospital rates go down same for death. But still about 1/3 are delta cases because 9/10 of Omicron infected do not go for a test...

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