Covid-19 News

  • Oh give me a break. That, like, never happens. You are saying I -- of all people -- do not give out information. Really? I have given out 4.8 million papers on cold fusion.

    What you say makes no sense. I have been saying "I don't know, I can't tell, I have not read enough, I am neutral on ivermectin." You demanding I give you proof that I don't know. Are you asking for a list of papers I have not read? That's nuts.

    I am sure you are aware there is a controversy. Okay, you are interested in this controversy. I am not. So you can sort out the controversy yourself, without help from me

    many people have died from Covid that didn't have to die because people like you peddle the crap coming from the CDC, NIH, and the FDA. You sir are the problem , vaccine, vaccine vaccine, no early intervention, your modern medicine is assbackwards and you promote propaganda.

  • These vaccines cannot alter our DNA:

    Robert answered already 90%. 10% of the rest problem is the RNA matching basic immune system that may trigger a change in methylation of DNA. That way the function of the cell gets blocked or activated.

    This is a modern brand of genetics called EPI genetics and is far more important than classic genetics.

    With one Pfizer jab you get between 30-50% pure junk RNA (because it decays fast. Factory quality = 70%) so millions of different epigenetic processes can potentially be triggered.

    I will never take the actual junk RNA vaccines. May be in 30 years they can do it better.

  • An evolutionary portrait of the progenitor SARS-CoV-2 and its dominant offshoots in COVID-19 pandemic…93/molbev/msab118/6257226


    Global sequencing of hundreds of thousands of genomes of Severe acute respiratory syndrome coronavirus 2, SARS-CoV-2, has continued to reveal new genetic variants that are the key to unraveling its early evolutionary history and tracking its global spread over time. Here, we present the heretofore cryptic mutational history and spatiotemporal dynamics of SARS-CoV-2 from an analysis of thousands of high-quality genomes. We report the likely most recent common ancestor of SARS-CoV-2, reconstructed through a novel application and advancement of computational methods initially developed to infer the mutational history of tumor cells in a patient. This progenitor genome differs from genomes of the first coronaviruses sampled in China by three variants, implying that none of the earliest patients represent the index case or gave rise to all the human infections. However, multiple coronavirus infections in China and the USA harbored the progenitor genetic fingerprint in January 2020 and later, suggesting that the progenitor was spreading worldwide months before and after the first reported cases of COVID-19 in China. Mutations of the progenitor and its offshoots have produced many dominant coronavirus strains, which have spread episodically over time. Fingerprinting based on common mutations reveals that the same coronavirus lineage has dominated North America for most of the pandemic in 2020. There have been multiple replacements of predominant coronavirus strains in Europe and Asia and the continued presence of multiple high-frequency strains in Asia and North America. We have developed a continually updating dashboard of global evolution and spatiotemporal trends of SARS-CoV-2 spread (

    The problem with this study is they never used samples from new York that showed Covid in august of 2019 and spain samples from march of 2019 both showing traces of the original SARS Cov2 virus genome . Covid had to be circulating in late 2018

  • COVID Patient in Coma Gets Ivermectin After Court Order…rmectin-after-court-order

    May 6, 2021 -- A 68-year-old woman with COVID-19, who has been in intensive care in an Illinois hospital for a month, started receiving the controversial drug ivermectin (Stromectol) this week after her family sued the hospital to have someone administer it, according to a report in the Chicago Tribune.

    Nurije Fype's daughter, Desareta, filed suit against Elmhurst Hospital, part of Edward-Elmhurst Health, asking that her mother receive the treatment, which is approved as an anti-parasite drug but not approved for the treatment of COVID-19. Desareta Fype has been granted temporary guardianship of her mother.

    The FDA has warned against ivermectin's use for treating COVID-19, but a high-profile group of doctors has spoken passionately in favor of it.

    The FDA has published guidance titled ,"Why You Should Not Use Ivermectin to Treat or Prevent COVID-19" on its website. The National Institutes of Health said there is not enough data to recommend either for or against its use in treating COVID-19.

    On Friday, DuPage County Judge James Orel ruled Fype should be allowed to get the treatment.

    Three days later, according to the Daily Herald, the lawyer for the hospital, Joseph Monahan, argued the hospital could not find a hospital-affiliated doctor to administer the ivermectin.

    The Herald reported the judge told the hospital to "get out of the way" and allow any board-certified doctor to administer the drug.

    When Fype's doctor was unable to administer it, the legal team found another doctor, Alan Bain, DO, to do it. Monahan said Bain was granted credentials to work at the hospital so he could administer it Monday evening.

    In a follow-up hearing on Tuesday, Monahan told Orel that the hospital asked 20 doctors and 19 other health care workers, including nurses and pharmacists, to administer the medication and they all declined, the Herald reported.

    Orel denied a request from Desareta Fype's lawyer to order the hospital's nurses to administer further doses. The judge also denied a request to hold the hospital in contempt of court.

    According to the Herald, the judge said at the hearing Tuesday, "Now let's hope the good Lord allows the medication to work."

    The hospital did not respond to Medscape's request for comment on the legal case or the patient's condition by publication time.

    According to NBC5 Chicago, Desareta said Tuesday, "She looks calm, comfortable, and I'm happy with her monitor numbers so far. They are kind of stable."

    Fype's daughter said her mother has been a patient at Elmhurst Hospital since April 7, was put on a ventilator on April 28, and is now in a coma.

    According to the Daily Herald, Desareta Fype said in court documents that she is willing to release the hospital from liability if the drug harms her mother.

    The Daily Herald also reports that Fype's attorneys cited arguments by the Front Line COVID Critical Care Alliance that ivermectin has antiviral and anti-inflammatory benefits that help people infected with COVID-19.

    Ivermectin is an anthelmintic used mainly to treat roundworms, threadworms, and other parasites.

    Medscape Medical News

  • How SARS-CoV-2 first adapted in humans

    Viruses need entry proteins to penetrate the cells where they will replicate. The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) version is called the spike or S protein. The S protein, also the target of the current vaccines, is quickly adapting to its new human hosts. It took its first major step in this direction early in 2020, when its amino acid 614 (of 1297) changed from an aspartic acid (D) to a glycine (G). Viruses bearing this D614G mutation transmit among humans more rapidly and now form the majority in circulation. On page 525 of this issue, Zhang et al. (1) use careful structural analyses to reveal how D614G changed the S protein to accelerate the pandemic.

    Early in the pandemic, in the scramble to create tools to study SARS-CoV-2, investigators developed pseudovirus systems to measure infection in a safe, easily quantifiable way. These systems express a viral entry protein on the surface of a reporter virus used to monitor cell entry and have been used for years to study many such proteins, including the S protein of “classic” SARS-CoV-1. Frustratingly, pseudoviruses built from the SARS-CoV-2 S protein produced much lower signals than those based on the very similar SARS-CoV-1 S protein. This was perplexing because biochemical studies of SARS-CoV-1 and SARS-CoV-2 S-protein receptor binding domains (RBDs) made clear that the SARS-CoV-2 RBD bound their common receptor, angiotensin-converting enzyme 2 (ACE2), with higher affinity than that of SARS-CoV-1 (2, 3). Faced with inefficient assays, many virologists landed on the same solution as their structural biology colleagues: Mutate the S-protein site that is cleaved by furin-like proteases in virus-producing cells (2). This change kept the S-protein S1 domain, which contains the RBD and binds ACE2, covalently linked to its S2 domain, which anchors the S protein to the virion. Notably, some—but not all—of these furin-site mutations significantly improved pseudovirus infection of cells (4).

    This fix solved a technical problem, but it deepened a mystery. Although a number of distantly related coronaviruses carry furin cleavage sites at their S1-S2 boundaries, the SARS-CoV-1 S protein, and those of all known bat-derived viruses from the same Sarbecovirus lineage, lack this site. Instead of being cleaved in virus-producing cells, their S proteins are cleaved by different proteases while the virus is engaging ACE2 in the next, yet-to-be-infected cell (5). As it happened, furin-site mutations that improved SARS-CoV-2 S-protein function in pseudoviruses allowed the modified S protein to work with these later-stage enzymes, just like the SARS-CoV-1 version. Why then did the SARS-CoV-2 furin site persist, even though it made infection in cell culture less efficient? Indeed, viruses passaged in culture regularly lost this site. Does it somehow improve viral transmission? Would it eventually disappear over the course of the pandemic?

    In the summer of 2020, Korber et al. sounded an alarm about a “mutation of concern,” namely D614G (6). In the laboratory, this change obviated the need to eliminate the S-protein furin site, apparently correcting a design flaw associated with this unusual cleavage site (4, 6). Animal studies with otherwise identical viruses showed markedly greater replication of the D614G variant in the upper respiratory tract, a site important for transmission (7, 8). By contrast, no significant differences between the two viruses were seen in the lower respiratory tract, a site responsible for more severe disease (7). These observations are consistent with the current consensus that D614G, now present in most circulating viruses, enhances viral transmission, but unlike more recent acquired mutations in S1 [e.g., Asn501→Tyr (N501Y)], it does not change the rate of hospitalizations.

    The underlying mechanism for this fitness advantage remained a point of controversy. Here, a second unusual property of the S protein, in this case shared with SARS-CoV-1, became relevant. The SARS-CoV-2 S protein, like most entry proteins of viruses with a lipid membrane, assembles into trimers. Typically, during the process of virion assembly, viral entry proteins subtly change their conformations, but it is unusual for these proteins to break their three-fold symmetry before they bind their receptor. However, the mature SARS-CoV-2 S protein often assumes an asymmetrical arrangement whereby one of its three RBDs assumes an open or “up” conformation (1, 9). Only RBDs in this up conformation can bind ACE2. Once it does so, the S1 domains dissociate from S2, and S2 undergoes a pronounced rearrangement to a “postfusion” state. The released energy of this rearrangement drives the viral and cell membranes to fuse and gives the virus access to the cell interior.

    To explain D614G fitness, some investigators focused on the impact of D614G on the frequency with which this one-up conformation could be found, suggesting that more efficient engagement of the receptor accounted for the enhanced transmissibility of viruses bearing this mutation (10, 11). Others noted that S proteins of D614G-expressing viruses fell apart less frequently, an effect perhaps amplified in the challenging environment of a living organism. They observed that D614G helped the S1 domain cling to S2, preventing S2 from prematurely and unproductively assuming its postfusion conformation (4, 9, 12). Thus, the virus had more functional S proteins that could bind and infect the next cell

    To cut through this controversy, Zhang et al. solved the structure and provided detailed analyses of both D614 and G614 S proteins in multiple states. They first noted that, as they and others had previously observed, the loss of D614 in S1 breaks an ionic bond to a proximal lysine, K854, in S2 (9). Loss of this salt bridge is initially counterintuitive because it would loosen the association between S1 and S2, although it might ease the movement of the RBD into the up configuration. However, the structures from Zhang et al. show that a major difference between S proteins with and without D614G is the visibly greater ordering in G614 S proteins of a region spanning residues 620 to 640, which the authors call the “630 loop.” This loop is just downstream of G614. It is therefore possible that either the loss of the D614-K854 salt bridge, or the greater backbone flexibility that a glycine affords, helps the 630 loop nestle more tightly in a canyon formed by two larger S-protein domains (the amino-terminal domain and carboxyl-terminal domain 1). Regardless, this loop is found in a more rigid and stable arrangement between these domains when residue 614 is a G than when it is a D.

    The key is that both the RBD-up conformation and dissociation of S1 from S2—enabled by furin cleavage—require disordering of the 630 loop. Thus, the RBD-up conformation can be more easily accessed with the original D614 S protein, but once this conformation is achieved, this S protein is more likely to fall apart entirely owing to premature shedding of its S1 domain. Conversely, with G614, more energy is required to achieve a one RBD–up state, but dissociation of S1 from S2 also becomes less favorable because the remaining folded 630 loops continue to hold the trimer together. Thus, the D614G variants have more S proteins in the up orientation because the next, irreversible step toward inactivation is slower. Infection with D614G is more efficient because it prevents premature S1 shedding (see the figure).

    These structural studies have real-life implications. All current vaccines are based on the original, unstable D614 form of the S protein (13). Fortunately, most vaccine developers, including Moderna and Pfizer-BioNTech, took a lesson from studies of SARS-CoV-1 and Middle East respiratory syndrome (MERS) coronavirus to slow S-protein shedding by introducing non-native prolines into S2 (14). Those who developed the Johnson and Johnson and Novavax vaccines had the prescience to also remove the furin site. By contrast, the developers of the University of Oxford–AstraZeneca vaccine opted for the wild-type S protein (containing D614), as is also the case for the inactivated virus vaccine produced by Sinovac. To be clear, other variables, especially antigen-delivery systems, likely account for efficacy differences among these vaccines. However, apples-to-apples studies in animals make clear that both engineered prolines and furin-site ablation contribute to vaccine effectiveness (15). It is almost certain that the next round of vaccines, those better reflecting the S-protein variants now in circulation, will include D614G. Vaccines that express unmodified S proteins with G614 may enjoy a relative jump in potency because this change compensates for the lack of engineered stabilizing mutations.

    The work of Zhang et al. also reveals more about the natural history of the virus. The notable emergence of D614G suggests that the acquisition of a destabilizing furin site was a recent event. The virus could easily lose this site, as it does frequently in cell culture systems, implying that it in some way facilitates human transmission. This is not a conclusion that most students of human coronaviruses would have anticipated, given that SARS-CoV-1, which transmits with reasonable efficiency, lacks this site, whereas the more distantly related MERS coronavirus bears this site and transmits poorly. How the SARS-CoV-2 furin site promotes new human infections remains a key open question in the field.

  • In a follow-up hearing on Tuesday, Monahan told Orel that the hospital asked 20 doctors and 19 other health care workers, including nurses and pharmacists, to administer the medication and they all declined, the Herald reported.

    This shows how well organized the pharma mafia in the US already is. Just to remind you. It worked the same way in XXXXX.

    Ivermectin is an anthelmintic used mainly to treat roundworms, threadworms, and other parasites.

    This is just one fairy tale how the mafia cheats simple minded people. Ivermectin so far is the cheapest, best working, broad band antiviral with no side effects at all. Remdesivir, Tamiflu just is dirty crap in view of Ivermectin.

    Ivermectin stopped the Zikka epidemic in Brazil, it works against flu (please remembers this, don't buy other bullshit anti flu drugs), Westnil virus, AIDS and many other virus. And Ivermectin works as a 100% preventive for SARS CoV-2 if you weekly take a normal dose.

    For Horses,Dogs, Cows Ivermectin is used as an antihelmic drug. But all worms carry a payload of virus, also many bacteria protect themselves with a viral payload. Thus only a broad band drug can eliminate a complex organism.

  • Not moot in Zimbabwe.. which is far way from 'we' in the US

    I meant that we should supply Zimbabwe with vaccines as soon as possible, rather than ivermectin or any other drug. Give everyone the one thing we know for sure works. Perhaps as a stopgap some other drugs might be used, but the best stopgap by far is to wear masks, social distance, track cases and so on. There is no doubt this works.

  • many people have died from Covid that didn't have to die because people like you peddle the crap coming from the CDC, NIH, and the FDA.

    Incorrect. The CDC, NIH and FDA are telling the truth. They are experts, and they have proven right by the data showing that vaccines eliminated the disease in Israel and elsewhere. Over the last 20 years they made it possible to develop mRNA vaccines, so they saved ~100 million lives. This is the greatest triumph in the history of public health.

    Many people have died because of anti-science, anti-vaccine movements -- the Death Cult fanatics who want to kill millions to enhance their own power. Many people have died because of lunatic opposition to masks and case tracking. In the U.S., hundreds of thousands more people died than necessary. This was entirely the fault of Trump and the Death Cult. They alone are to blame. If we had listened to CDC, WHO and others, this would not have happened.

    Your views are based on false conspiracy theories, not facts, and not science.

    You sir are the problem ,

    No, the Death Cult is the problem. They are to blame for everything. Look at countries like Korea and Japan and you will see that is obvious. They never employed ivermectin or any other untested drug, but their cases and deaths per capita were far lower than ours. They used mainly tried-and-true public health measure, such as masks and case tracking.

  • Various questions that have arisen here have clarified in recent news.

    In Florida, at least one major cruise ship line wants vaccine passports, but the governor has forbidden them. So, it is industry and customers who want them, as I thought.…ine-florida-desantis.html

    Cruise Line Threatens to Skip Florida Ports Over Proof-of-Vaccination Ban

    Norwegian Cruise Line plans to require Covid-19 vaccine documentation from its crew members and customers, but Florida recently enacted a law that bars businesses from doing so.

    We now know why cases in the Seychelles are increasing, despite the fact that a large fraction of the population was vaccinated. They are using Chinese vaccines. Apparently these do not work well.…les-vaccines-covid-cases/

    The number of deaths in the U.S. is probably far higher than the ~600,000 on record, according to an estimate based on excess deaths in 2020. That is what you would expect for the early months of the pandemic, before there was adequate testing.…-reported-covid-19-deaths

  • I meant that we should supply Zimbabwe with vaccines as soon as possible, rather than ivermectin or any other drug.

    Can somebody stop this free mason mafioso? Zimbabwe has no need at all for vaccines. There are a few cases each day.

    But Zimbabwe destroys the free masons picture as they brought down CoV-19 without vaccines. Only Ivermectin

    Japan and you will see that is obvious. They never employed ivermectin or any other untested drug,

    Japan is heavily using Ivermectin. One more country the free mason are going to fail. My wive is Japanese and has a bit more insight than the old globe trotter...

    The number of deaths in the U.S. is probably far higher than the ~600,000 on record, according to an estimate based on excess deaths in 2020.

    Bullshit. The US counts deaths as Switzerland does. Even people shot do death that within 30 days before were PCR positive are counted as Covid deaths. This is from an US friend - not free mason...

    In reality it's the other way round. May be JED complains that some cancer patients that missed the treatment due to CoV-19 did not contain the virus (in the hospital as most did) and so should be counted too...

  • Tucker Carlson on FOX recently gave an informative talk on the cause for concern.

    For instance : VAERS (self reporting system for adverse vaccine reactions) shows roughly 200 people dying per year after the flu vaccine.

    The same VAERS shows roughly 3500 people dying this year after a Covid vaccine.

    Video here:

    So what should my decision be? I do not take this lightly.

    My decision is to be a volunteer for the unvaccinated Control Group for the current Covid vaccine Experiment.

    Although not getting a vaccine is apparently dangerous, I'll make that sacrifice ... for science!


  • Important Sunday reading for jed rothwell,

    Immediate Use of Ivermectin Medicine Globally Can End COVID-19 Pandemic: Scientists…can-end-covid-19-pandemic

    A peer-reviewed research has claimed that global ivermectin use can end the COVID-19 pandemic, as the medicine significantly reduces the risk of contracting the deadly respiratory disease when used regularly.

    The common antiparasitic ivermectin is being touted as a miracle cure for COVID-19 by doctors and campaigners the world over.

    Peer reviewed by medical experts that included three US government senior scientists and published in the American Journal of Therapeutics, the research is the most comprehensive review of the available data taken from clinical, in vitro, animal, and real-world studies.

    Led by the Front Line COVID-19 Critical Care Alliance (FLCCC), a group of medical and scientific experts reviewed published peer-reviewed studies, manuscripts, expert meta-analyses, and epidemiological analyses of regions with ivermectin distribution efforts all showing that ivermectin is an effective prophylaxis and treatment for COVID-19.

    "We did the work that the medical authorities failed to do, we conducted the most comprehensive review of the available data on ivermectin," said Pierre Kory, MD, president and chief medical officer of the FLCCC. "We applied the gold standard to qualify the data reviewed before concluding that ivermectin can end this pandemic."

    A focus of the manuscript was on the 27 controlled trials available in January 2021, 15 of which were randomised controlled trials (RCT's).

    Consistent with numerous meta-analyses of ivermectin RCT's since published by expert panels from the UK, Italy, Spain and Japan, they found a large, statistically significant reduction in mortality, time to recovery, and viral clearance in COVID-19 patients treated with ivermectin.

    "Our latest research shows, once again, that when the totality of the evidence is examined, there is no doubt that ivermectin is highly effective as a safe prophylaxis and treatment for COVID-19," said Paul E. Marik, founding member of the FLCCC and Chief, Pulmonary and Critical Care Medicine at Eastern Virginia Medical School.

    Many regions around the world now recognise that ivermectin is a powerful prophylaxis and treatment for COVID-19. South Africa, Zimbabwe, Slovakia, Czech Republic, Mexico, and India have approved the drug for use by medical professionals.

    The results, as seen in this latest study, demonstrate that the ivermectin distribution campaigns repeatedly led to "rapid population-wide decreases in morbidity and mortality".

    "We are calling on regional public health authorities and medical professionals around the world to demand that ivermectin be included in their standard of care right away so we can end this pandemic once and for all," Marik noted.

  • In fight against COVID variants some firms target T cell jabs…ants-firms-cell-jabs.html

    Getting COVID vaccines into the arms of the world's population is an international priority—but will today's jabs stay effective against virus variants that are spreading across the globe?

    It is one of the big questions about the pandemic, with Pfizer chief Albert Bourla recently acknowledging that it is likely a booster will be needed to help extend the protection conferred by its vaccine and ward off new variants.

    A recent study presented a mixed picture.

    It found that the antibody response of current vaccines could fail against variants. However, a second immune response in the form of killer T cells—which attack already infected cells and not the virus itself—remained largely intact.

    Several startups are working on developing shots centred on T cells in hopes of producing a jab that would not only provide protection against new virus strains already on the loose, but also variants that don't yet exist.

    Alexis Peyroles heads up French biotech firm OSE Immunotherapeutics, which is developing a vaccine that targets T cells that has just begun clinical trials.

    "It could offer several years of protection," he told AFP.

    Another French firm, Lyon-based Osivax, is also working on a T cell shot, promising a "universal" vaccine that would be effective against any potential variant.

    The government of France, which has yet to develop its own vaccine, is supporting the effort with millions in funding.

    Such projects are far from widespread. Among the 400 vaccines under development counted by the World Health Organization only a few are aimed at universal use.

    The most advanced shot of its kind is the ImmunityBio vaccine under development in the United States. Very preliminary results released last month were mostly encouraging.

    'Complement and broaden'

    No lab foresees a final product before next year and many scientists are sceptical about the usefulness of trying to develop a shot to protect against a virus strain that doesn't yet exist.

    "Mass vaccination itself is a form of evolutionary 'selection' pressure," British virologist Julian Tang told AFP, "and this pressure may push the virus to evolve to escape any vaccine protection—so it can be a double-edged sword."

    Other questions involve the extent to which the body will be able to fight the virus with a T cell-based response.

    T cells and antibodies work together to form an immune response in the body.

    French virologist Yves Gaudin pointed out that if an antibody response fails, "T cells don't serve much purpose".

    He said he is "doubtful about the effectiveness of such a vaccine," emphasising that an ideal vaccine would be effective in both areas.

    In Europe and the United States the plan for T cell jabs, should they see the light of day, would be to give them to people who had already received the current antibody vaccines.

    Peyroles confirmed that OSE's vaccine, should it prove effective in trials, is indeed meant as a way to strengthen current inoculations.

    "You would complement and broaden the response created by the first vaccines in terms of scope and time."

    He added that T cell vaccines could offer protection to people who have difficulties developing antibodies due to other ailments such as diabetes or cancer.

  • This is how dishonest national media promote false truths . Florida has basically declared the pandemic under control and this is how the national media responds.

    Florida reports more than 10,000 COVID-19 variant cases, surge after spring break

    243 have been hospitalized with variants and 67 have died.…s-surge/story?id=77553100

    Variant COVID-19 infections skyrocketed following spring break in Florida and there have been more than 10,000 variant cases reported throughout the state, the South Florida Sun Sentinel reported based on data from the Florida Department of Health.

    A total of 753 variant cases from three strains -- the B.1.1.7, the P.1, and the B. -- were reported on March 14, according to variant infection data shared with ABC News. The Florida Department of Health does not disclose variant cases on its public dashboard.

    That number swelled to 5,177 cases from five types of variants on April 15. Just two weeks later, the number of variant infections exploded to 9,248 on April 27, according to local ABC affiliate , WFTV.

    The surge falls in line with mid-March into April spring break celebrations, when college students and vacationers flock to the sunshine state.

    Florida is home to the most variant COVID-19 cases in the country. State health officials reported more than 11,800 cases of COVID-19 variants on Wednesday, according to the Sun Sentinel.

    In total, variants have led to the hospitalization of 243 residents and the death of 67 people in Florida, the Sun Sentinel reported.

    Only 1% of all COVID-19 cases in Florida undergo testing to study their genetic coding, meaning the number of variant infections is likely much higher than reported.

    The data regarding variants was first released Monday hours after Gov. Ron DeSantis announced the lift of all local COVID-19 restrictions.

    Now the reality

    3,977 new Florida coronavirus cases reported Saturday; 65 new deaths…aturday-65-new-deaths.amp

    Fear mongering continues and confusion rules!

  • COVID-19 variant in India may be evading vaccine protection, top WHO scientist says…/who-india-covid-variant/

    GENEVA – A COVID-19 variant spreading in India is more contagious and may be dodging vaccine protections, contributing to the country’s explosive outbreak, the World Health Organization’s chief scientist said Saturday.

    In an interview, Soumya Swaminathan warned that “the epidemiological features that we see in India today do indicate that it’s an extremely rapidly spreading variant.”

    India on Saturday for the first time registered more than 4,000 COVID-19 deaths in just 24 hours, and more than 400,000 new infections.

    New Delhi has struggled to contain the outbreak, which has overwhelmed its health care system, and many experts suspect the official death and case numbers are a gross underestimate.

    Swaminathan, an Indian pediatrician and clinical scientist, said the B.1.617 variant of COVID-19, which was first detected in India last October, was clearly a contributing factor to the catastrophe unfolding in her homeland.

    “There have been many accelerators that are fed into this,” the 62-year-old said, stressing that “a more rapidly spreading virus is one of them.”

    The WHO recently listed B.1.617 — which counts several sublineages with slightly different mutations and characteristics — as a “variant of interest.”

    But so far it has stopped short of adding it to its short list of “variant of concern” — a label indicating it is more dangerous than the original version of the virus by being more transmissible, deadly or able to get past vaccine protections.

    Several national health authorities, including in the United States and Britain, have meanwhile said they consider B.1.617 a variant of concern, and Swaminathan said she expected the WHO to soon follow suit.

    “B 1.617 is likely to be a variant of concern because it has some mutations which increase transmission, and which also potentially could make (it) resistant to antibodies that are generated by vaccination or by natural infection,” she said.

    But she insisted that the variant alone could not be blamed for the dramatic surge in cases and deaths seen in India, lamenting that the country appeared to have let down its guard down, with “huge social mixing and large gatherings.”

    Mass election rallies held by Prime Minister Narendra Modi and other politicians have for instance partly been blamed for the staggering rise in infections.

    But even as many in India felt the crisis was over, dropping mask-wearing and other protection measures, the virus was quietly spreading.

    “In a large country like India, you could have transmission at low levels, which is what happened for many months,” Swaminathan said.

    “It was endemic (and) probably gradually increasing,” she said, decrying that “those early signs were missed until it reached the point at which it was taking off vertically.”

    “At that point it’s very hard to suppress, because it’s then involving tens of thousands of people and it’s multiplying at a rate at which it’s very difficult to stop.”

    While India is now trying to scale up vaccination to rein in the outbreak, Swaminathan warned that the jabs alone would not be enough to gain control of the situation.

    She pointed out that India, the world’s largest vaccine-making nation, had only fully vaccinated around 2% of the 1.3 billion-plus population.

    “It’s going to take many months if not years to get to the point of 70% to 80% coverage,” she said.

    With that prospect, Swaminathan stressed that “for the foreseeable future, we need to depend on our tried and tested public health and social measures” to bring down transmission.

    The surge in India is frightening not only due to the horrifying number of people who are sick and dying there, but also because the exploding infection numbers dramatically increase the chances of new and more dangerous variants emerging.

    “The more the virus is replicating and spreading and transmitting, the more chances are that … mutations will develop and adapt,” Swaminathan said.

    “Variants which accumulate a lot of mutations may ultimately become resistant to the current vaccines that we have,” she warned.

    “That’s going to be a problem for the whole world.”

  • In fight against COVID variants some firms target T cell jabs

    What about a soft infection + Ivremectin that does the same. All at more or less no cost?

    While India is now trying to scale up vaccination to rein in the outbreak, Swaminathan warned that the jabs alone would not be enough to gain control of the situation.

    And this idiot blocks Ivermectin on state level...Why do people elect natural born killers as leaders?

  • Frontline Doctors Launch National RV Tour to Combat Covid-19 Medical Censorship and Cancel Culture

    America's Frontline Doctors is Bringing "The Uncensored Truth Tour" to Cities Across America to Help Physicians and Patients Stand Up for Science, Freedom, and Common Sense…el-culture-301286789.html

    LOS ANGELES, May 7, 2021 /PRNewswire/ -- As millions of Americans continue to deal with the impacts of government overreach and the politicized science surrounding COVID-19, America's Frontline Doctors (AFLDS) is launching a nationwide RV tour to help patients make informed decisions about their care. The tour, entitled The Uncensored Truth: Physicians and Patients Standing Up for Science, Freedom and Common Sense, will kick off on Monday May 10 with events in Phoenix, Arizona.

    At each stop AFLDS physicians and attorneys will engage local communities in a critical conversation about healthcare, the law, and how all Americans can protect both themselves and their freedom.

    Throughout the pandemic, AFLDS has been a powerful voice for doctors and patients who have been misinformed, victimized and silenced by public health bureaucrats, the media and politicians. Reaching millions of Americans each month with unbiased information about COVID-19 healthcare options, AFLDS has been at the forefront of the national conversation about treatment, prevention and effective, common sense approaches to COVID that don't infringe on civil liberties.

    AFLDS physicians have been the victims of cancel culture for the past year for raising objections to the government's response to the pandemic. Today, both the facts and the science about a range of issues surrounding COVID-19 have come to support their positions.

    "Big Tech, Big Media, and Big Government love to play doctor, but only your chosen physician should be involved in your private healthcare decisions, and no one should be silencing front-line first responders," said AFLDS founder Dr. Simone Gold, who will be traveling and meeting with physicians and the public at each stop.

    "Doctors, nurses, business owners and patients across the country have been intimidated by politicians and public health bureaucrats for wanting alternatives to dangerous lockdowns, unproven Big Pharma vaccines, and restrictions on freedom that are not based on science. We will not be cancelled. This tour will help advance that critical conversation and empower people with information," continued Dr. Gold. Like many Americans, AFLDS supports the appropriate use of FDA-approved vaccines and opposes COVID-19 vaccine mandates or passports being contemplated by government and private sector entities.

    "At every turn, AFLDS has been a consistent and credible voice for transparency, evidence-based science, the ethical practice of medicine, and common sense," stated Dr Shelley Cole, Medical Director for AFLDS. "The Uncensored Truth tour will help encourage Americans who feel silenced and give them the facts to make informed healthcare decisions for themselves and their families."

    The first leg of The Uncensored Truth tour across the southern U.S. will include:

    Phoenix AZ

    May 10

    Albuquerque NM

    May 11

    Lubbock TX

    May 12

    Dallas TX

    May 13

    Austin TX

    May 14

    San Antonio TX

    May 15

    Houston TX

    May 16

    New Orleans LA

    May 17

    Tallahassee FL

    May 18

    Naples FL

    May 22

    Miami FL

    May 23

    Tampa FL

    May 23

    Orlando FL

    May 24

    Palm Coast FL

    May 25

    Jacksonville FL

    May 26

    Those interested in attending a AFLDS event in their areas can learn more and register at


    To book an AFLDS member physician for your media outlet or program, please send requests to [email protected] . America's Frontline Doctors (AFLDS) is a non-partisan, not-for-profit organization. AFLDS stands up for every American looking for the best quality healthcare by empowering doctors working on the front lines of our nation's most pressing healthcare challenges. Our growing community of member physicians come from across the country representing a range of medical disciplines and practical experience. To learn more about America's Frontline Doctors, visit


    Bob Driscoll

    [email protected]


    SOURCE America's Frontline Doctors (AFLDS)

  • This is a bombshell!!!!!

    Chinese scientists discussed weaponising SARS coronaviruses 5 years before Covid pandemic: Report…_articleshow/82498904.cms

    BEIJING: A document written by Chinese scientists and health officials before the pandemic in 2015 states that SARS coronaviruses were a "new era of genetic weapons" that could be "artificially manipulated into an emerging human disease virus, then weaponised and unleashed, reported Weekend Australian.

    The paper titled The Unnatural Origin of SARS and New Species of Man-Made Viruses as Genetic Bioweapons suggested that World War Three would be fought with biological weapons. The document revealed that Chinese military scientists were discussing the weaponisation of SARS coronaviruses five years before the Covid-19 pandemic.

    Peter Jennings, the executive director of the Australian Strategic Policy Institute (ASPI), told that the document is as close to a "smoking gun" as we have got.

    "I think this is significant because it clearly shows that Chinese scientists were thinking about military application for different strains of the coronavirus and thinking about how it could be deployed," Jennings said.

    "It begins to firm up the possibility that what we have here is the accidental release of a pathogen for military use," Jennings added.

    He also said that the document may explain why China has been so reluctant for outside investigations into the origins of Covid-19

    "If this was a case of transmission from a wet market it would be in China's interest to co-operate ... we've had the opposite of that."

    Robert Potter, a cyber security specialist who analyses leaked Chinese government documents, was asked by The Australian to verify the paper. He says the document definitely isn't fake, reported

    "We reached a high confidence conclusion that it was genuine ... It's not fake but it's up to someone else to interpret how serious it is," Potter said.

    It emerged in the last few years ... they (China) will almost certainly try to remove it now it's been covered."

    Potter further stated that it isn't unusual to see Chinese research papers discussing areas that they're behind on and need to make progress in.

    "It's a really interesting article to show what their scientific researchers are thinking," he added.

    The Covid-19 pandemic has been caused by a coronavirus named SARS-CoV-2 which emerged in December 2019. Coronaviruses are a large family of viruses, several of which cause respiratory diseases in humans - ranging from a common cold to Severe Acute Respiratory Syndrome (SARS).

    Since the Covid-19 pandemic began, there have been over 157 million cases of infections and 3.28 million deaths worldwide, according to the latest update by Johns Hopkins University.