Covid-19 News

Quote from JedRothwell

All of the studies of animals in cages separated by masks shows this, with every kind of mask. All of the physical tests and simulations show this.

As said already, you are an illiterate and never did read any study about aerosols. What you cite is droplets studies. Flu usually leads to long lasting cough where droplets are the main issue.

All countries with outdoor masks here in Europe have 3x death from CoV-19 than Switzerland without. These idiots did read the same papers as you and obviously did kill some 10'000 people!!

I'm happy that Swiss politicians have a little more brain and also read actual papers. Also Swiss children are happy that they can attend primary/secondary school without masks.

Quote from Mark U

In the coming weeks, CDC will transition from monitoring all reported vaccine breakthrough cases to focus on identifying and investigating only vaccine breakthrough infections that result in hospitalization or death.

This is illegal marketing support for Pfizer/Moderna as it is now more than obvious that the efficiency of the vaccines declines over time. But what else do you expect from a FM/R/J group conspiracy.??

We so far had at least 18 death of fully vaccinated people most of them older than 80 years. This again is just the tip of the iceberg. Still no counting of the 2 weeks immune deficiency killed people.

Quote from Mark U

Before May 2021, if you tested positive and had been vaccinated, you were directed to have the "COVID-19 Vaccine Breakthrough Case Investigation Form" form filled out.

https://www.cdc.gov/vaccines/c…se-Investigation-Form.pdf

But now you have to enter hospital and/or die to have the form filled out. We can no longer tell what number of the reported positive cases were vaccinated or unvaccinated. Here is the CDC's excuse for this :

In the coming weeks, CDC will transition from monitoring all reported vaccine breakthrough cases to focus on identifying and investigating only vaccine breakthrough infections that result in hospitalization or death. This shift will help maximize the quality of the data collected on cases of greatest clinical and public health importance.

They're saying that lowing the quantity of forms filled out will maximize the quality of the data.

I suppose what they are saying is that forms filled out by doctors are more reliable than forms filled out by patients. It is a short form, so I doubt any doctor would hesitate to fill it out. But, in any case, all cases are reported to the CDC, breakthrough or not.

Quote from Mark U

Of course, the VAERS database has naught to do with 'vaccine breakthrough' cases.

Surely it does. It is a database of adverse effects from the vaccine. A breakthrough case is about as adverse as you can get, other than death from the vaccine. In any case, the CDC is keeping track of all cases reported by doctors or patients. They are not investigating mild breakthrough cases in as much depth as they did earlier this year.

Quote from Mark U

Incidentally, one has to marvel over the new term 'vaccine breakthrough' that has replaced the traditional term 'vaccine failure'.

The term "breakthrough case" was coined in 1941 and it become common in the 1980s. See:

https://books.google.com/ngram…eakthrough%20case%3B%2Cc0

I suppose it is more exact than "vaccine failure" because there are other ways a vaccine can fail.

Quote from JedRothwell

Surely it does. It is a database of adverse effects from the vaccine.

Yes it is. But an adverse effect from the vaccine itself is very different than an adverse effect from the disease that the vaccine is supposed to prevent.

Quote from JedRothwell

The term "breakthrough case" was coined in 1941 and it become common in the 1980s. See:

https://books.google.com/ngram…eakthrough%20case%3B%2Cc0

"Breakthrough case" could refer to almost anything. Try "vaccine breakthrough" :

Search for "Vaccine Breakthrough" yielded only one result.

"Breakthrough infection" yields results though. But "vaccine breakthrough", in the context of a disease that has eluded a vaccine's protection, that's a pretty new one.

I don't think this will surprise many here

Hospitals may be overcounting how many kids are admitted for COVID-19 in the US, study finds

https://amp.usatoday.com/amp/5073193001

A new study found health care providers may be overcounting the number of kids hospitalized for COVID-19, overestimating the small impact the disease has on children

Researchers at the Stanford University School of Medicine analyzed COVID-19 data from Lucile Packard Children’s Hospital Stanford from May 10, 2020, to Feb. 10. During the nine-month period, 117 patients under the age of 18 either tested positive for SARS-CoV-2 at the hospital or were hospitalized for multisystem inflammatory syndrome in children, or MIS-C.

Out of the 117 children, nearly 40% of COVID-19 cases were asymptomatic, according to the study published Wednesday in the peer-reviewed journal Hospital Pediatrics. About 45% of those hospital admissions were categorized as unlikely to be caused by the virus.

“It’s in keeping with what other studies have shown, which is that children in general are relatively mildly affected by the infection,” said Dr. Asim Ahmed, a pediatric infectious disease specialist unaffiliated with the study and senior medical director at Karius, an infectious disease diagnostic company.

Dr. Alan Schroeder, study co-author and clinical professor of pediatric critical care and pediatric hospital medicine at Stanford, said it’s important to distinguish between children who test positive but are asymptomatic and those who are hospitalized for COVID-19 to understand how the disease truly affects the pediatric population.

“Our goal is to make sure we have accurate data on how sick children are getting,” he said. “If we rely on hospitals’ positive SARS-CoV-2 test results, we are inflating by about twofold the actual risk of hospitalization from the disease in kids.”

The study found about 28% had mild to moderate COVID-19, 7.7% had severe illness, 12.8% had critical illness and 12% of patients had MIS-C, a serious condition that appears to be connected to COVID-19 in which some organs and tissues can become severely inflamed.

Study authors said it’s likely many children develop other conditions that require hospitalization, then test positive for SARS-CoV-2 without knowing they were infected. Though this data may be useful to better understand community transmission, it doesn’t accurately measure the rate at which kids get sick with COVID-19, said Dr. Roshni Mathew, lead author and clinical associate professor of pediatric infectious diseases at Stanford.

Just knowing that a child is hospitalized and has the virus is not enough information to determine if they are actually sick with COVID-19,” she said.

Researchers said some of the study’s limitations may have affected its results. For example, the study sample size is relatively small compared with other regions of the country where COVID-19 may have been more prevalent.

Data may be skewed by the unusually low hospitalization rates at pediatric centers, Ahmed said. Those were partly due to parents’ reluctance to take their kids to a hospital for fear they could get sick and the low prevalence of illnesses, such as the flu, after public health measures were implemented to control the pandemic.

Just knowing that a child is hospitalized and has the virus is not enough information to determine if they are actually sick with COVID-19,” she said.

Researchers said some of the study’s limitations may have affected its results. For example, the study sample size is relatively small compared with other regions of the country where COVID-19 may have been more prevalent.

Data may be skewed by the unusually low hospitalization rates at pediatric centers, Ahmed said. Those were partly due to parents’ reluctance to take their kids to a hospital for fear they could get sick and the low prevalence of illnesses, such as the flu, after public health measures were implemented to control the pandemic.

Even though the hospitalization patterns and practices were a certain way for the adult hospitals (during the pandemic), they were very different for the pediatric hospitals,” he said. “The census across the board for pediatric hospitals was quite low.”

Severe disease is rare among children with COVID-19, but health experts still urge parents to get their kids vaccinated as severe disease, hospitalizations and deaths do occur.

About 300 children have died from COVID-19 in the USA, said Dr. John Williams, chief of the division of pediatric infectious diseases at UPMC Children’s Hospital of Pittsburgh.

He said getting children vaccinated is important to prevent coronavirus transmission and protect people who are immunocompromised and may not respond as robustly to the COVID-19 vaccines.

It’s an important study, and it’s an area that we need to keep learning about,” he said. “But it should not be interpreted to say that children don’t need vaccines."

Quote from Mark U

"Breakthrough case" could refer to almost anything. Try "vaccine breakthrough" :

Try it yourself. It has the same pattern as "breakthrough case" starting in 1941:

https://books.google.com/ngram…ne%20breakthrough%3B%2Cc0

Ngram indicates that "breakthrough case" is more common than "vaccine breakthrough." Probably, the latter more often describes an important advance in vaccines, rather than a breakthrough case (a vaccine failure).

The CDC describes "vaccine breakthrough infection" and "breakthrough infection" but I do not see "vaccine breakthrough" by itself not followed by "infection." Ngram cannot handle "vaccine breakthrough infection."

Google's linguistics AI is good at sorting out context and filtering out coincidental appearances of "breakthrough" and "case" appearing together in a non-medical context. "The police made a breakthrough in the case" would probably not register. Making a breakthrough in a police investigation surely predates 1941, but Ngram shows nothing before 1941. "Breakthrough" was coined before 1800.

Google AI is pretty good, but I should point out that Google translate from English to Japanese failed all three: "breakthrough case" and "breakthrough infection" "vaccine breakthrough infection." Google translate has many of the AI software tools used by Ngram.

Quote from Fm1

A new study found health care providers may be overcounting the number of kids hospitalized for COVID-19, overestimating the small impact the disease has on children

Researchers at the Stanford University School of Medicine analyzed COVID-19 data from Lucile Packard Children’s Hospital Stanford from May 10, 2020, to Feb. 10. During the nine-month period, 117 patients under the age of 18 either tested positive for SARS-CoV-2 at the hospital or were hospitalized for multisystem inflammatory syndrome in children, or MIS-C.

This seems unlikely to me. Perhaps there were some in May and June 2020, but soon after that, tests became widely available. I am sure they tested everyone suspected of having COVID-19 after July. I doubt there were many false positives. They would probably test two or more times with a pediatric case. Any pediatric case that came into the hospital was serious, and called for careful monitoring. It would be a big problem if they misdiagnosed it.

Quote from Mark U

Yes it is. But an adverse effect from the vaccine itself is very different than an adverse effect from the disease that the vaccine is supposed to prevent.

We are talking about breakthrough cases, where the vaccine failed to prevent the disease. That is definitely an adverse effect from the vaccine. Okay, if you want to nitpick, it is a lack of positive effect. The vaccine did not create enough antibodies. I am 100% sure it merits inclusion in VAERS, and probably other databases.

Enormous attention was paid to breakthrough cases at first, because they had to establish the efficacy of the vaccines; confirm the double blind test infection rate; and determine how likely it was that a patient with breakthrough cases would infect others. It turned out to be very unlikely. That's a relief, because the cases are so mild, some patients don't know they are sick.

Quote from JedRothwell

I am 100% sure it merits inclusion in VAERS, and probably other databases.

How can you be so sure, when it isn't true? Check out

https://vaers.hhs.gov/resources/infoproviders.html

and

https://vaers.hhs.gov/docs/VAE…Following_Vaccination.pdf

You will see that one doesn't report to VAERS the disease the vaccine is supposed to prevent.

There is a type of exception however. This occurs when an attenuated, live virus vaccine produces the disease in the vaccine recipient. Such was the case for the live polio oral vaccine - which occasionally produced polio or polio like symptoms in those receiving the vaccine.

Quote from JedRothwell

Enormous attention was paid to breakthrough cases at first, because they had to establish the efficacy of the vaccines; confirm the double blind test infection rate; and determine how likely it was that a patient with breakthrough cases would infect others. It turned out to be very unlikely.

You're talking as if this is all in the past and is all figured out now. It isn't! This continues to be an ongoing experiment. We don't really know how readily breakthrough cases can infect others. For instance, from

https://ca.finance.yahoo.com/n…ted-player-215734826.html

we see that nine breakthrough cases are now reported in the New York Yankees organization. They are quarantining those breakthrough cases. So, they don't seem very confident that breakthrough cases are 'very unlikely' to transmit the virus, even to the vaccinated.

For those who want to hear in 20 minutes what is wrong in North America about how we have (not) treated Covid outpatients, here's a very worthwhile video from March of 2021, showing Dr. Peter McCullough testifying before the Texas Senate Health and Human Services committee.

A New, Infectious Coronavirus Is Detected In Malaysia – And It's Coming From Dogs

https://www.npr.org/sections/g…and-it-s-coming-from-dogs

In the past 20 years, new coronaviruses have emerged from animals with remarkable regularity. In 2002, SARS-CoV jumped from civets into people. Ten years later, MERS emerged from camels. Then in 2019, SARS-CoV-2 began to spread around the world.

For many scientists, this pattern points to a disturbing trend: Coronavirus outbreaks aren't rare events — but will likely occur every decade or so.

Now, scientists are reporting that they have discovered what may be the latest coronavirus to jump from animals into people. And it comes from a surprising source: dogs.

When the COVID-19 pandemic first exploded, Dr. Gregory Gray started to wonder if there might be other coronaviruses out there, already making people sick and threatening to trigger another outbreak.

The problem was, he didn't have a tool to look for them. The test for COVID-19, he says, is extremely limited. It tells whether one particular virus – SARS-CoV-2 – is present in a person's respiratory tract, and nothing else.

"Diagnostics are very specific. They generally focus on known viruses," says Gray, an infectious disease epidemiologist at Duke University's Global Health Institute.

So he challenged a graduate student in his lab, Leshan Xiu, to make a more powerful test — one that would work like a COVID-19 test but could detect all coronaviruses, even the unknown ones.

Xiu not only rose to the challenge, but the tool he created worked better than expected.

In the first batch of samples tested last year, Gray and Xiu found evidence of an entirely new coronavirus associated with pneumonia in hospitalized patients — mostly in kids. This virus may be the eighth coronavirus known to cause disease in people, the team reports Thursday in the journal Clinical Infectious Diseases.

The samples came from patients at a hospital in Sarawak, Malaysia, taken by a collaborator in 2017 and 2018. "These were deep nasal swabs, like doctors collect with the COVID-19 patients," says Gray.

The patients had what looked like regular pneumonia. But in eight out of 301 samples tested, or 2.7%, Xui and Gray found that the patients' upper respiratory tracts were infected with a new canine coronavirus — a dog virus.

"That's a pretty high prevalence of a [new] virus," Gray says. "That's remarkable." So remarkable, in fact, that Gray actually thought maybe he and Xiu had made a mistake. Perhaps Xiu's test wasn't working quite right. "You always wonder if there was a problem in the lab," he says.

To find out, he sent the patients' samples over to a world expert on animal coronaviruses at Ohio State University. She was also dubious. "I thought, there's something wrong," says virologist Anastasia Vlasova. "Canine coronaviruses were not thought to be transmitted to people. It's never been reported before."

Nevertheless, Vlasova went to work. She tried to grow the coronavirus in the lab, using a special solution she knew worked for other dog coronaviruses. Lo and behold, "the virus grew very well," she says.

With a lot of virus on hand, Vlasova could decode its genome. From the virus' gene sequences, she could see that the virus had likely infected cats and pigs at one point. But it likely jumped directly from dogs into people. "The majority of the genome was canine coronavirus," she says.

Then she found a disturbing clue about the virus' future. "We did discover a very, very unique mutation — or deletion — in the genome," Vlasova says. That specific deletion, she says, isn't present in any other known dog coronaviruses, but it is found somewhere else: in human coronaviruses. "It's a mutation that's very similar to one previously found in the SARS coronavirus and in [versions of] SARS-CoV-2 ... [that appeared] very soon after its introduction into the human population," Vlastova says.

This deletion, she believes, helps the dog virus infect or persist inside humans. And that it may be a key step required for coronaviruses to make the jump into people.

"Apparently the deletion is somehow associated with [the virus'] adaptation during this jump from animal to human," she says.

Altogether, this genetic data suggest that Vlasova and her colleagues are catching this new coronavirus early on in its journey in people, while it's still trying to figure out how to infect people efficiently — and possibly, before it can spread from person to person and trigger a big outbreak.

There's no evidence yet of transmission from human to human," says virologist Xumin Zhang at University of Arkansas for Medical Sciences. But it's not known how these patients became infected with the virus, or if they had direct contact with infected animals.

Zhang has studied coronaviruses for more than 30 years. He thinks it's too early to call this new virus a human pathogen. "As the authors are careful to say in their paper, they have not proven what's called Koch's postulates," he says. That is, Vlasova, Gray and colleagues haven't shown that the new coronavirus causes pneumonia; so far, it's only been associated with the disease. "To do that, strictly, they need to inject the virus into humans and see if it reproduces the disease," he says. "Of course [for ethical reasons], we cannot do that." Instead, Zhang says, they can look to see how common the virus is in pneumonia patients around the world — and they can test to see if it makes mice or another animal sick.

Yet Zhang says he wouldn't be surprised if this dog virus is, in fact, a new human pathogen. He thinks the more scientists look for unknown coronaviruses inside pneumonia patients, the more they are going to find. "I believe there are many animal [coronaviruses] out there that can transmit to humans."

And in order to stop a future coronavirus pandemic, he says, scientists need to do more testing in people and seek out these strange, hidden infections — before they become a problem.

a little more to previous post

Two more coronaviruses can infect people, studies suggest

https://www.sciencemag.org/new…ct-people-studies-suggest

Eight children hospitalized with pneumonia in Malaysia several years ago had evidence of infections with a novel coronavirus similar to one found in dogs, a research team reports today. Only seven coronaviruses were previously known to infect people, the latest being SARS-CoV-2, the spark of the COVID-19 pandemic. The discovery of this likely new human pathogen, along with the report of an instance of a coronavirus that appears to have jumped from pigs to people many years ago, could significantly expand which members of the viral family pose another global threat.

“I think the more we look, the more we will find that these coronaviruses are crossing species everywhere,” says Stanley Perlman, a virologist at the University of Iowa who was not involved in the new work.

The researchers have not conclusivey linked either new virus to human disease. And there’s no evidence that the two new coronaviruses can transmit between people—each infection may have been a dead-end jump into a person from nonhuman host. But many researchers worry the viruses may evolve that ability within a person or the animals they normally infect. A complete genome sequence of the virus found in one Malaysian patient, reported today in Clinical Infectious Diseases, reveals a chimera of genes from four coronaviruses: two previously identified canine coronaviruses, one known to infect cats, and what looks like a pig virus.

This is the first report suggesting a caninelike coronavirus can replicate in people, and further studies will need to confirm the ability. The researchers have grown the virus in dog tumor cells but not yet in human cells.

Unlike with SARS-CoV-2 and other known human coronaviruses, “We don’t have any clear evidence that this particular [coronavirus] strain is better adapted to humans because of its spike structure,” says veterinary virologist Anastasia Vlasova of the Ohio State University (OSU), lead author of the study. Human infections from dog coronaviruses may occur “at a much higher frequency than we previously thought,” she adds. This particular virus might not transmit between people, but we don’t know that for sure, Vlasova cautions.

The eight children whose tissue samples Vlasova and her colleagues studied were mainly living in traditional longhouses or villages in rural or suburban Sarawak on Borneo, where they likely had frequent exposure to domestic animals and jungle wildlife. They were among 301 hospitalized pneumonia patients during 2017–18 and the researchers screened each patient's nasopharyngeal samples—tissue swabbed from the upper part of the throat—for a large variety of human and nonhuman coronaviruses.

Standard hospital diagnostics for pneumonia or other respiratory illness would not have detected dog and cat coronaviruses. No one has been looking for these viruses in patients with such illnesses until recently. “These canine and feline coronaviruses are everywhere in the world,” Perlman says.

The entire novel virus sequence from the children’s samples most resembles a canine coronavirus. However, the sequence for its spike protein, which attaches to host cell receptors to initiate an infection, is closely related to the spike sequence of canine coronavirus type I and the one for a porcine coronavirus known as transmissible gastroenteritis virus (TGEV). And one part of the spike protein bears a 97% similarity to the spike of a feline coronavirus.

This chimera is unlikely to have arisen at once, but instead involved repeat genetic reshuffles between different coronaviruses over time. “This is a mosaic of several different recombinations, happening over and over, when nobody’s watching. And then boom, you get this monstrosity,” says virologist Benjamin Neuman at Texas A&M University.

The animal that actually transmitted the novel virus to the people could have been a cat, pig, dog, “or some wild carnivores,” says Vito Martella, a veterinary virologist at the University of Bari in Italy. He plans to screen stored fecal samples from Italian children with acute gastroenteritis to see if he can find something similar.

Researchers already knew that three canine coronavirus subtypes mix readily with feline and porcine coronaviruses. “What is more surprising is that these [animal] viruses can actually cause disease in a person,” Perlman says, because one would expect them to lack some of the genes important for adapting well to people.

Seven of the eight children whose tissues harbored sequences of the virus were younger than 5 years old, and four of them were infants, mostly from Indigenous ethnic groups. Each was hospitalized for 4 to 7 days and recovered.

Scientists divide coronaviruses into four genera—alpha, beta, gamma, and delta—and the new one is an alpha. It is the third such alpha coronavirus to infect people; the other two cause common colds, and most people are exposed to them early in life. That pattern may explain why only children were perhaps sickened by this new one. Ralph Baric, a virologist at the University of North Carolina, Chapel Hill, suggests adults may have some immunity to the newly discovered alpha coronavirus due to repeated exposure to the other two.

So far, the most dangerous human coronaviruses—SARS-CoV-1, SARS-CoV-2 and MERS-CoV—are the betas. Researchers haven’t seen alphas trigger an outbreak of serious disease in humans, Neuman says, “but that doesn’t feel like much comfort in the wild world of viruses.”

In March, researchers at the University of Florida reported in a medRxiv preprint the first evidence of a porcine delta coronavirus that infects people, in serum from 3 Haitian children who had fevers in 2014—15. The researchers transferred serum samples into monkey cells and were able to grow viruses that they matched, genetically, to known porcine coronaviruses. (The work has been submitted to a peer-reviewed journal.)

Delta coronaviruses were once thought to infect only birds. Then, in 2012, a delta-coronavirus infected swine in Hong Kong. It “appears to have jumped over from songbirds,” says OSU coronavirologist Linda Saif, who went on to isolate the virus in swine cell cultures.

The same virus caused a major fatal diarrheal disease outbreak in baby pigs in the United States in 2014. It has since been shown to infect cell lines from humans, pigs, and chickens; lab studies have shown that the virus causes persistent infection and diarrheal disease when put into poultry. “It’s out on its own, a left-field-type virus that infects both avian and mammalian species,” Baric says. “There aren’t any other coronaviruses that I know can do this.”

Some virologists have labeled the Hong Kong delta coronvirus a pandemic threat. The Haitian virus differs considerably and virologists want to test local children and adults for antibodies to it. If its ability to infect people is confirmed, it may also be viewed as a pandemic threat, Saif says.

Together, the two reports point to the importance of animal diseases in public health, and the need for coronavirus vaccines for domesticated animals. “This research clearly shows that more studies are desperately needed to evaluate critical questions regarding the frequency of cross-species [coronavirus] transmission and potential for human-to-human spread,” Baric says.

Gregory Gray at Duke University, the senior author on the Malaysian chimeric coronavirus study, also advocates for surveillance among pneumonia patients in areas known to be hot spots for novel viruses or places where large populations of animals and humans mix, such as live animal markets and large farms. “These spill overs take years,” Gray says. “It’s not like in the movies. They go through different steps to infect humans.”

A pilot-randomized, double-blind crossover trial to evaluate the pharmacokinetics of orally administered 25-hydroxyvitamin D3 and vitamin D3 in healthy adults with differing BMI and in adults with intestinal malabsorption

https://academic.oup.com/ajcn/…0?redirectedFrom=fulltext

ABSTRACT

Background

Obese and malabsorptive patients have difficulty increasing serum 25-hydroxyvitamin D [25(OH)D] after taking vitamin D supplementation. Since 25(OH)D is more hydrophilic than vitamin D, we hypothesized that oral 25(OH)D supplementation is more effective in increasing serum 25(OH)D concentrations in these patients.

Objectives

We aimed to investigate the pharmacokinetics of oral 25-hydroxyvitamin D3 [25(OH)D3] and oral vitamin D3 in healthy participants with differing BMI and malabsorptive patients.

Methods

A randomized, double-blind crossover trial was performed in 6 malabsorptive patients and 10 healthy participants who were given 900 µg of either vitamin D3 or 25(OH)D3 orally followed by a pharmacokinetic study (PKS). After ≥28 d from the first dosing, each participant returned to receive the other form of vitamin D and undergo another PKS. For each PKS, serum vitamin D3 and 25(OH)D3 were measured at baseline and at 2, 4, 6, 8, and 12 h and days 1, 2, 3, 7, and 14. Pharmacokinetic parameters were calculated.

Results

Data were expressed as means ± SEMs. The PKS of 900 µg vitamin D3 revealed that malabsorptive patients had 64% lower AUC than healthy participants (1177 ± 425 vs. 3258 ± 496 ng · h/mL; P < 0.05). AUCs of 900 µg 25(OH)D3 were not significantly different between the 2 groups (P = 0.540). The 10 healthy participants were ranked by BMI and categorized into higher/lower BMI groups (5/group). The PKS of 900 µg vitamin D3 showed that the higher BMI group had 53% lower AUC than the lower BMI group (2089 ± 490 vs. 4427 ± 313 ng · h/mL; P < 0.05), whereas AUCs of 900 µg 25(OH)D3 were not significantly different between the 2 groups (P = 0.500).

Conclusions

Oral 25(OH)D3 may be a good choice for managing vitamin D deficiency in malabsorption and obesity. This trial was registered at clinicaltrials.gov as (NCT03401541.

How Covid 19 made 9, Vaccine billionares!

COVID vaccines mint new billionaires as BioNTech and Moderna CEOs' wealth soars

https://uk.finance.yahoo.com/n…2hIylseWfyXhQdicJxU_u60d9

The nine new vaccine billionaires:

Stéphane Bancel — Moderna’s CEO (worth $4.3bn)

Ugur Sahin, CEO and co-founder of BioNTech (worth $4bn)

Timothy Springer — an immunologist and founding investor of Moderna (worth $2.2bn)

Noubar Afeyan — Moderna’s Chairman (worth $1.9bn)

Juan Lopez-Belmonte — Chairman of ROVI, a company with a deal to manufacture and package the Moderna vaccine (worth $1.8bn)

Robert Langer — a scientist and founding investor in Moderna (worth $1.6bn)

Zhu Tao co-founder and chief scientific officer at CanSino Biologics (worth $1.3bn)

Qiu Dongxu, co-founder and senior vice president at CanSino Biologics (worth $1.2bn)

Mao Huinhoa, also co-founder and senior vice-president at CanSino Biologics (worth $1bn)

In addition, the combined wealth of eight existing billionaires — who have extensive portfolios in the COVID-19 vaccine firms — increased by $32.2bn, according to the Alliance.

Quote from Fm1

How Covid 19 made 9, Vaccine billionares!

COVID vaccines mint new billionaires as BioNTech and Moderna CEOs' wealth soars

https://uk.finance.yahoo.com/n…2hIylseWfyXhQdicJxU_u60d9

The nine new vaccine billionaires:

Stéphane Bancel — Moderna’s CEO (worth $4.3bn)

Ugur Sahin, CEO and co-founder of BioNTech (worth $4bn)

Timothy Springer — an immunologist and founding investor of Moderna (worth $2.2bn)

Noubar Afeyan — Moderna’s Chairman (worth $1.9bn)

Juan Lopez-Belmonte — Chairman of ROVI, a company with a deal to manufacture and package the Moderna vaccine (worth $1.8bn)

Robert Langer — a scientist and founding investor in Moderna (worth $1.6bn)

Zhu Tao co-founder and chief scientific officer at CanSino Biologics (worth $1.3bn)

Qiu Dongxu, co-founder and senior vice president at CanSino Biologics (worth $1.2bn)

Mao Huinhoa, also co-founder and senior vice-president at CanSino Biologics (worth $1bn)

In addition, the combined wealth of eight existing billionaires — who have extensive portfolios in the COVID-19 vaccine firms — increased by $32.2bn, according to the Alliance.

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FM,

Does it say how much $ they had before Covid-19 hit?

Quote from Roseland67

FM,

Does it say how much $ they had before Covid-19 hit?

Good question I'll try to dig that up

Quote from Roseland67

FM,

Does it say how much $ they had before Covid-19 hit?

After a short search, short being the keyword it looks like all were or on their way to billionaire status but this might help to put some perspective to the article. Also remember, it was your tax dollars going to develope and manufacture these vaccines. When should I expect my dividend check from moderns and Pfizer-BioNTech.

Three Billionaires Behind Pfizer's Vaccine Partner BioNTech Are $2 Billion Richer After It Announces Covid-19 Vaccine Results

https://www.forbes.com/sites/g…-results/?sh=11a625234784

Jeff Bezos, Elon Musk among US billionaires getting richer during coronavirus pandemic

https://www.usatoday.com/story…er-during-covid/43205617/

Another associate dies from blood clot.

A second (in three weeks) person I have some contact with has "mysteriously" died of a blood clot.

I did not know this person or his family well, so I would not intrude by asking private information.

What I do know is :

early 40's

In apparent good health, non-smoker, not over weight, etc.

Almost assuredly had Covid vaccinations relatively recently, but I do not know which kind or exactly when.

Completely unexpected, i.e. no accident with broken bones etc. Daily life and then bang.

I know this is anecdotal and proves nothing. But I tried hard and could not recollect a single case of anyone I knew dying of blood clots with out having an injury. (I did know a lady who had a bicycle wreck and broke her leg. She developed a clot that broke loose to her heart several days later and was fatal)

The fact that two have now crossed my path and the completely benign circumstances around them other than the current Covid vaccinations makes me wonder........

Quote from Bob#2

Another associate dies from blood clot.

A second (in three weeks) person I have some contact with has "mysteriously" died of a blood clot.

I did not know this person or his family well, so I would not intrude by asking private information.

What I do know is :

early 40's

In apparent good health, non-smoker, not over weight, etc.

Almost assuredly had Covid vaccinations relatively recently, but I do not know which kind or exactly when.

Completely unexpected, i.e. no accident with broken bones etc. Daily life and then bang.

I know this is anecdotal and proves nothing. But I tried hard and could not recollect a single case of anyone I knew dying of blood clots with out having an injury. (I did know a lady who had a bicycle wreck and broke her leg. She developed a clot that broke loose to her heart several days later and was fatal)

The fact that two have now crossed my path and the completely benign circumstances around them other than the current Covid vaccinations makes me wonder........

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It is easy for .2% of the population to start dying from the vaccine and nobody will figure it out for a year.

Thus, the vaccine is equivalent to the disease.

Quote from Fm1

Out of the 117 children, nearly 40% of COVID-19 cases were asymptomatic, according to the study published Wednesday in the peer-reviewed journal Hospital Pediatrics. About 45% of those hospital admissions were categorized as unlikely to be caused by the virus.

The big pharma mafia (FM/R/J) want's everybody as a customer. Only damaged humans that blindly follow your order are good customers.

Vaccination of anybody younger than 35 - without any real medical reason - is a crime.

Europe just decided to go the fascists way (eugenics path). More rights (Freedom to travel) for gene manipulated people only. The base we all believed to live on just now disappears.

Next steps after Covid-pass: AIDS-pass, Flu-pass, Hepatitis-pass, measles pass...