There are somewhat less risky vaccines (Janssen?) and even better there are the combined approaches of diet/lifestyle, vitamin supplements and Ivermectin.
As I'm am not a Guinea pig I will not take a RNA vaccine. The Pfizer vaccine simply is here thanks to multiple fraud. This is the main reason not to touch it. J&J is very save for males age >60. RNA vaccines arn't safe for any group. You take it at your own risk...
As I'm am not a Guinea pig I will not take a RNA vaccine. The Pfizer vaccine simply is here thanks to multiple fraud. This is the main reason not to touch it. J&J is very save for males age >60. RNA vaccines arn't safe for any group. You take it at your own risk...
Tight variable but these statistics give an overwhelming conclusion that besides J&J and perhaps up and comers, less expensive drugs and vitamin C/D will do your virus in well. Not many want to admit it, but it seems a lot more clear. There do seem to be significant sex differences in response to vaccine types and treatments.
Dr Surya Kant
The story of how his Chess club saved thousands of lives in
the state of Uttar Pradesh , the sixth largest 'country' in the world
TM 25.15
Q. "The WHO recently advised against ivermectin...? Any comments?
TM 25.30
A. " I am convinced that this drug can save my life
this is my moral responsibility as an Indian citizen of this country,
I have to protect these lives of my people
so I will use ivermectin
Whatever WHO says? I will never mind.
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Three Wuhan lab researchers were hospitalized in November 2019: report
https://nypost.com/2021/05/23/…november-2019-report/amp/
Three researchers at a Chinese lab that has been scrutinized as the possible origin point of the coronavirus pandemic went to the hospital due to an illness in November 2019, according to a new report.
The Wall Street Journal, which cited current and former US officials, reported that the intelligence gathered by “an international partner” expands on a State Department document confirming that workers at the Wuhan Institute of Virology fell ill with symptoms “consistent with both Covid-19 and common seasonal illness” in fall 2019.
The officials were split on the strength of the intelligence, with one telling the Journal it needed more corroboration and another saying it was “of exquisite quality” and “very precise.” Both agreed that the intelligence stopped short of confirming the researchers had contracted coronavirus.
Many health experts believed that the coronavirus began circulating in the central Chinese city of Wuhan in November 2019. Authorities in Beijing date the first confirmed case to Dec. 8 of that year.
The theory that the coronavirus accidentally leaked from the Wuhan Institute of Virology into the wider population has gained credence as a viable explanation in recent weeks, following a World Health Organization-led investigation and report — compiled with the help of the Chinese government — that left many other nations dissatisfied
China has contributed to the suspicion, with the lab not releasing records related to its work on coronaviruses in bats. Meanwhile, Beijing has pushed a series of wild theories, including that the coronavirus spread through imported frozen food packaging and originated at a biomedical research facility at Fort Detrick in Maryland.
Earlier this month, a group of top scientists from around the globe pushed back on the WHO’s conclusion that a lab-leak is “extremely unlikely” — arguing in the journal Science that theory is “viable” and deserves “a proper investigation.”
Meanwhile, Republicans are pressing federal health officials to detail what the Wuhan Institute of Virology did with grant money from the National Institutes of Health NIH.
On Friday, six GOP senators and one congressman signed a letter to NIH Director Dr. Francis Collins requesting details about a 2014 moratorium on funding so-called “gain of function research” — in which viruses are manipulated in a lab environment to become more transmissible and harmful. The letter also requested more information about the lifting of the moratorium in late 2017 and whether programs and researchers linked to the Wuhan lab were granted exceptions.
White House chief medical adviser Dr. Anthony Fauci has repeatedly denied that the NIH funded gain of function research in Wuhan, but Sen. Tom Cotton (R-Ark.) accused Fauci of playing “word games” Sunday.
“The money that the NIH gave went to an American organization which turned around and gave hundreds of thousands of dollars to these Wuhan labs to investigate coronaviruses and yes, to find ways to make them more contagious and more dangerous,” Cotton told Fox Business Network’s “Sunday Morning Futures”.
“I think that there could be an example here of these public health bureaucrats thinking they know better, that they’re not going to answer to political oversight and accountability … and they went ahead with this research that could be very dangerous,” Cotton added. “That’s why it is imperative that the NIH come clean and tell us exactly what happened.”
New Human Coronavirus CCoV-HuPn-2018 Identified – Originated in Dogs
https://scitechdaily.com/new-h…d-originated-in-dogs/amp/
Discovery shows need to monitor animal viruses, researchers say.
Researchers have identified and completed the genetic analysis of a newly discovered coronavirus – one that has evolved from a coronavirus that afflicts dogs to infect humans and may contribute to respiratory symptoms.
The discovery of the first dog coronavirus found to have crossed over to infecting people underscores the treacherous nature of coronaviruses and the need to monitor animal viruses as a way of predicting possible threats to public health, researchers say.
“At this point, we don’t see any reasons to expect another pandemic from this virus, but I can’t say that’s never going to be a concern in the future,” said Anastasia Vlasova, an assistant professor in The Ohio State University College of Food, Agricultural, and Environmental Sciences (CFAES).
She conducted the study with Gregory C. Gray, a professor in the Division of Infectious Diseases of the Duke University School of Medicine, and Teck-Hock Toh, a professor at SEGi University in Sarawak, Malaysia.
In 2018, researchers analyzed the nasal swabs of 301 patients treated in an East Malaysian hospital for pneumonia. Eight patients, all but one of them children, were found to have been infected with the newly discovered coronavirus that the study’s researchers named CCoV-HuPn-2018. All eight patients were treated and released after four to six days in the hospital, where they were given oxygen to help them breathe.
In a study published on May 20, 2021, in the journal Clinical Infectious Diseases, researchers describe the genetic characteristics of CCoV-HuPn-2018, suggesting it’s a new coronavirus that moved from infecting dogs to infecting people.
We don’t really have evidence right now that this virus can cause severe illness in adults,” Vlasova said, citing the fact that only one person in the study found to have been infected with the new coronavirus was an adult. “I cannot rule out the possibility that at some point this new coronavirus will become a prevalent human pathogen. Once a coronavirus is able to infect a human, all bets are off.”
Viruses change constantly. When a virus alters its genetic makeup enough to go from infecting only a certain type of animal to infecting people, a combination of factors determine how well the new virus can replicate and spread among people.
For an animal coronavirus to infect people, the virus must first enter the human body and recognize something on the surface of the cells, then bind to those cells.
“We know this virus can do that,” Vlasova said.
However, that transmission from a dog to a person may be a dead end for CCoV-HuPn-2018 if the virus does not replicate well once inside the person, or if the person’s immune system fends it off, Vlasova said.
Only about half of the genetic makeup of the newly discovered coronavirus is similar to that of the SARS-CoV-2 virus causing the COVID-19 pandemic, Vlasova said.
Although only eight patients in the study were found to have been infected with the CCoV-HuPn-2018 virus, the study’s researchers say that this or highly similar viruses likely have circulated much further than that among dogs and people in Malaysia.
“A one-time sampling is not going to tell you with accuracy how prevalent it is,” Vlasova said. “The sampling has to be repeated and done over a period time to see how many people become infected.”
If confirmed through further epidemiological studies, this new coronavirus could be the eighth coronavirus shown to trigger illnesses in people.
“If you had mentioned this 20 years ago, that a virus that affects dogs could change to be able to infect people, many would have been skeptical,” Vlasova said.
Even though this new coronavirus comes from a dog, it may not be necessary for people to change how they interact with their dogs in light of this study, Vlasova said.
“But I would definitely be watching a little more how much I allow my babies around dogs,” she said. Seven of the eight people hospitalized in Malaysia and found to be infected with the CCoV-HuPn-2018 virus were children, one as young as 5 1/2 months old.
The CCoV-HuPn-2018 virus has different symptoms from the dog virus it came from, which caused gastrointestinal problems in dogs such as diarrhea and upset stomach. People infected with the CCoV-HuPn-2018 virus experience a respiratory illness that does not include the gastrointestinal issues.
“We are likely missing important animal viruses that are beginning to adapt to humans,” Gray said. “We need to conduct such virus discovery work among people with pneumonia and also among people who have intense exposure to animals so that we get early warning of a new virus which may become a future pandemic virus.”
The potential threat posed by the viruses of dogs or cats, which also suffer illnesses from coronaviruses, has not been studied widely, though it should be, Vlasova said. Monitoring animal viruses is a way of protecting public health, she said.
“We primarily put the emphasis on studying emerging disease in humans – not animals,” Vlasova said. “That’s a big flaw in the current approach.”
Reference: “Novel Canine Coronavirus Isolated from a Hospitalized Pneumonia Patient, East Malaysia” by Anastasia N Vlasova, PhD, Annika Diaz, Debasu Damtie, MS, Leshan Xiu, MS, Teck-Hock Toh, MD, Jeffrey Soon-Yit Lee, MD, Linda J Saif, PhD, Gregory C Gray, MD, 20 May 2021, Clinical Infectious Diseases.
DOI: 10.1093/cid/ciab456
White House chief medical adviser Dr. Anthony Fauci has repeatedly denied that the NIH funded gain of function research in Wuhan, but Sen. Tom Cotton (R-Ark.) accused Fauci of playing “word games” Sunday.
It's grand time to jail the US Dr.Mengele Fauci for organizing mass murder.
New Human Coronavirus CCoV-HuPn-2018 Identified – Originated in Dogs
Why do the us media not follow up on the Italy cases from September 2019?????????
These cases (+ New York April 2019) document that it was an intentional release.
Highly Pathogenic Bird Flu Outbreak Already Reported in 46 Countries, Scientists Warn
https://www.sciencealert.com/h…opped-scientists-warn/amp
While the world was distracted with the rampant spread of a novel coronavirus, 2020 also witnessed an explosion in another deadly pathogen that could pose a threat to global public health.
H5N8, a subtype of highly pathogenic avian influenza virus (HPAIV), was identified decades ago, but during 2020 a series of emerging and ongoing H5N8 outbreaks in avian populations across dozens of countries have led to the death or slaughter of millions of birds worldwide.
"The affected geographic regions have been expanding continuously, and at least 46 countries have reported highly pathogenic H5N8 AIV outbreaks," virus researchers Weifeng Shi and George F. Gao write in a new perspective article in Science, warning of the dangers of H5N8 if we don't closely monitor and contain this worrisome trend.
While the most vulnerable animals to H5N8 are different kinds of birds (including farmed chicken and ducks, but also wild and migratory birds), human cases of the virus have also been discovered in recent times.
An outbreak of the avian flu in Russia in December 2020 jumped to poultry workers, with seven people on a farm in southern Russia showing signs of the infection – representing the first time H5N8 had ever been found in humans.
While that was a first for H5N8, it certainly wasn't a first for clades and subclades related to H5N8, nor for avian flu viruses in general
These cases were from 17 countries, with ~76 percent from Egypt and Indonesia."
But zoonotic risks are only part of the problem with H5N8 and its ilk. In most of the recent outbreaks, a clade of H5N8 called 2.3.4 has become the dominant pathogen worldwide, first seen in a Chinese wet market in 2010.
"Clade 2.3.4 H5 AIVs, particularly the H5N8 subtype, have clearly displayed a propensity for rapid global spread in migratory birds," the researchers write, noting that these viruses also display evidence of constant evolution, genetically reassorting themselves with parts of other AIV subtypes.
Shi and Gao – respectively from China's Shandong First Medical University and the Chinese Center for Disease Control and Prevention – were among some of the first scientists to document the novel coronavirus in early 2020.
They note that the subsequent COVID-19 pandemic – and the prevention and control measures world populations enacted in response – saw a sharp reduction in the spread of seasonal human influenza A and B viruses in the last year.
Nonetheless, in the same timeframe, a number of highly pathogenic H5Ny AIVs, including H5N1, H5N2, H5N5, and H5N8 subtypes, spread across China, South Africa, Europe, Eurasia, and elsewhere.
At the same time, research has shown that clade 2.3.4 viruses show particular cell-binding adaptations that could pose greater risks for human transmission, including potentially human-to-human transmissibility.
In all, the researchers say we need to significantly step up our surveillance of HPAIVs in poultry farms now, before these pathogens fly the coop.
"Because of the long-distance migration of wild birds, the innate capacity for reassortment of AIVs, the increased human-type receptor binding capability, and the constant antigenic variation of HPAIVs, it is imperative that the global spread and potential risk of H5N8 AIVs to poultry farming, avian wildlife, and global public health are not ignored," Shi and Gao write.
The findings are reported in Science.
It's grand time to jail the US Dr.Mengele Fauci for organizing mass murder.
Why do the us media not follow up on the Italy cases from September 2019?????????
These cases (+ New York April 2019) document that it was an intentional release.
The media along with our experts at the CDC and the NIH supposedly debunked the early testing finding Covid in Italy in September of 2019, New York was July/august, 2019 and Spain in March/April ,2019. Now that the lab leak and gain of function back on the table, these finding from 2019 must be reinvestigated.
Wyttenbach, the UK is taking wastewater treatment to the next level, this hopefully brings those early test results back to light.
Explained: How the UK is testing sewage to detect the presence of Covid-19
The UK government is expanding its testing of wastewater to detect the presence of coronavirus and its emerging variants. Why test wastewater, and how does it work?
https://indianexpress.com/arti…of-covid-19-7325752/lite/
The UK government is expanding its testing of wastewater to detect the presence of coronavirus and its emerging variants.
According to the country’s Department of Health and Social Care, the testing has now been ramped up to cover about two-thirds of the population. A new laboratory situated in Exeter, which will analyse the samples of wastewater, has now become one of the biggest such labs in the world.
Newsletter | Click to get the day’s best explainers in your inbox
Why test wastewater?
Over a year ago, researchers in Australia developed an early warning surveillance system to track the community transmission of COVID-19 by tracing the presence of the virus’s genes in raw sewage. This was possible because some infected individuals may shed the virus in their stool or urine.
This method is especially useful in identifying the scale of spread of the disease in a community. By detecting and quantifying the amount of virus found in the samples gives an indication of how widespread the virus is in an area, information that can be used by decision-makers to put restrictions on movement for certain people or start testing in an area more aggressively. Even in areas where widespread testing may not be feasible, adopting this method can point out the efficacy of containment measures.
For instance, wastewater testing in the Luton Borough Council of England led to the understanding that the increase in cases was not a result of community transmission, but limited to person-to-person transmission.
Specifically, in the UK researchers are now looking at the spread of variants including the one first detected in South Africa and India.
How does wastewater testing work?
After wastewater samples are taken from about 500 locations across the country, they are sent to the laboratory at Exeter Science Park. Here, scientists test and analyse the water samples to quantify the amount of virus present in them.
Further, some of these samples are sent to other laboratories where they are sequenced to identify variants.
Niclosamide: Beyond an antihelminthic drug
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5628105/
Abstract
Niclosamide is an oral antihelminthic drug used to treat parasitic infections in millions of people worldwide. However recent studies have indicated that niclosamide may have broad clinical applications for the treatment of diseases other than those caused by parasites. These diseases and symptoms may include cancer, bacterial and viral infection, metabolic diseases such as Type II diabetes, NASH and NAFLD, artery constriction, endometriosis, neuropathic pain, rheumatoid arthritis, sclerodermatous graft-versus-host disease, and systemic sclerosis. Among the underlying mechanisms associated with the drug actions of niclosamide are uncoupling of oxidative phosphorylation, and modulation of Wnt/β-catenin, mTORC1, STAT3, NF-κB and Notch signaling pathways. Here we provide a brief overview of the biological activities of niclosamide, its potential clinical applications, and its challenges for use as a new therapy for systemic diseases.
Keywords: Niclosamide, Cancer, Bacterial and viral infection, Lupus, Metabolic diseases, Type II diabetes, NASH, NAFLD, Neuropathic pain, Rheumatoid arthritis, Sclerodermatous graft-versus-host disease, Systemic sclerosis
A 'Game Changer' COVID-19 drug from Korea was introduced in a world class academic journal
-- A study on CNPharm's antiviral oral formulation published in a special edition of Pharmaceuticals
-- Hyundai Bioscience to proceed to clinical trial with CP-COV03
Wyttenbach, the UK is taking wastewater treatment to the next level,
One more time: This test has been developed at ETH EAWG (Zürich). It has also been used for early Milano testing. In Milano stored samples of waste water, the virus was present already in November 2019... So no debunking possible!
Q. "The WHO recently advised against ivermectin...? Any comments?
Tamil Nadu.. population 70 million .. stopped ivermectin rollout after 3 days and bought Remdesivir..
..political experiment..following the WHO advice... its nice to have a control group. except if you are in it in Tamil Nadu.
8 hrs ·
There is only one state in India not using {the "I" medicine} in their protocol. This state is Tamil Nadu.
This simple chart shows the difference between Tamil Nadu and the rest of the top 7 states by population. There is simply no question that {the "I" medicine} is the component that is saving tens of thousands of lives.
Thanks to Juan Chaime, data analyst, for the graph.
https://joannenova.com.au/2021…vermectin-use/#more-77662
Bangladesh also not minding the WHO.. using plenty of IVM..
deaths at 1/3 the rate of India (pop.=166 million)
Nice graph by Masud via flourish..
https://public.flourish.studio…ign=visualisation/5789739
Bangla is making progress .. used to be dirt poor..
a little silver in your mouthwash keeps Covid away
Evaluation of silver nanoparticles for the prevention of SARS-CoV-2 infection in health workers: in vitro and in vivo
https://www.medrxiv.org/conten…5.20.21256197v1.full-text
Abstract
SARS-CoV-2 infection in hospital areas is of a particular concern, since the close interaction between health care personnel and patients diagnosed with COVID-19, which allows virus to be easily spread between them and subsequently to their families and communities. Preventing SARS-CoV-2 infection among healthcare personnel is essential to reduce the frequency of infections and outbreaks during the pandemic considering that they work in high-risk areas. In this research, silver nanoparticles (AgNPs) were tested in vitro and shown to have an inhibitory effect on SARS-CoV-2 infection in cultured cells. Subsequently, we assess the effects of mouthwash and nose rinse with ARGOVIT® silver nanoparticles (AgNPs), in the prevention of SARS-CoV-2 contagion in health workers consider as high-risk group of acquiring the infection in the General Tijuana Hospital, Mexico, a hospital for the exclusive recruitment of patients diagnosed with COVID-19. We present a prospective randomized study of 231 participants that was carried out for 9 weeks (during the declaration of a pandemic). The “experimental” group was instructed to do mouthwash and nose rinse with the AgNPs solution; the “control” group was instructed to do mouthwashes and nose rinse in a conventional way. The incidence of SARS-CoV-2 infection was significantly lower in the “experimental” group (two participants of 114, 1.8%) compared to the “control” group (thirty-three participants of 117, 28.2%), with a 84.8% efficiency. We conclude that the mouth and nasal rinse with AgNPs helps in the prevention of SARS-CoV-2 infection in health personnel who are exposed to patients diagnosed with COVID-19.
Conclusions
This prospective randomized study demonstrates that mouthwash and nose rinse with AgNPs is effective in decreasing SARS-CoV-2 infection rate. To our knowledge, this study is the first experimental in vitro e in vivo trial where AgNPs as mouthwash and nasal rinse solution are applied for SARS-CoV-2 contagion prevention. The results of this investigation are revealing and encouraging, due to the fact that the health personnel exposed to an excessive viral load due to the number of COVID-19 patients attended (169 patients attended on average per week per person) were not infected, which is attributed to rinsing with AgNPs. Proved its inhibitory effect on SARS-CoV-2 infectivity in vitro it is inferred that the use of AgNPs as a mouthwash and nose rinse will be very useful as a prophylactic for the prevention of SARS-CoV-2, not only for health care personnel, but also as additional protection for the general population.
Tamil Nadu.. population 70 million .. stopped ivermectin rollout after 3 days and bought Remdesivir..
..political experiment..following the WHO advice... its nice to have a control group. except if you are in it in Tamil Nadu.
Cases in Tamil Nadu are skyrocketing. https://www.mygov.in/covid-19
How much bribe money did go into Tamil Nadu state officials pockets? The Gilead crap has almost no effect on CoV-19.
I agree: A control group is the best they could do and will convince the rest of the world.
This prospective randomized study demonstrates that mouthwash and nose rinse with AgNPs is effective in decreasing SARS-CoV-2 infection rate.
Also the Trump bleach (ClO2-X) included (low concentration) in a mouth wash is very effective but may be not political correct...
Washington U study finds clues to long-term COVID-19 immunity in survivors' bone marrow
https://www.stltoday.com/lifes…5a-a2d1-c01592280fc0.html
ST. LOUIS — Researchers here have discovered cells in the bone marrow of COVID-19 survivors that were still producing antibodies against the virus months after recovery, suggesting immunity may last far longer than once thought.
In a study released Monday in Nature, researchers show that among a group of 19 participants who previously had mild cases of COVID-19, 15 had antibody-producing cells in their bone marrow that target the virus that causes COVID-19.
“That’s strong evidence for long-lasting immunity,” said Ali Ellebedy, an associate professor at Washington University and senior author of the study.
The researchers took bone marrow samples from 18 participants seven to eight months after their initial infections. The cells were still found four months later in five participants who returned.
The researchers did not find the antibody-producing cells in a control group of 11 people who had never had the virus.
Ellebedy said Washington University researchers are also in the process of studying individuals who have been vaccinated.
The bone marrow work stemmed out of an ongoing study at Washington University, where researchers were tracking antibody levels in the blood of 77 participants, most of whom had mild cases of COVID-19. Their data showed antibody levels in the blood dropping quickly in the first few months of infection, then leveling, with still detectable amounts 11 months after infection.
The researchers acknowledged that the study only looked at the bone marrow of people who had mild cases of the virus, not those who had asymptomatic cases, or moderate or severe cases. And Ellebedy said that the age group of the participants was relatively young, so it is unclear whether an older group of individuals would have shown the same immune response. The participants ranged from 30 to 69 years old.
Dr. Sharon Frey, clinical director for St. Louis University’s Center for Vaccine Development, said scientists have suspected the presence of these cells, because they are found with other viruses, like influenza.
Still, said Frey, who was not involved in the study, the findings are positive, and exciting.
The body’s response to COVID-19 is complex, Frey said, and the cells are “just a piece of the overall immune response.” There are still many unknowns about how long the protection will last, and how protective they will be against different variants of the virus.
“It’s just a piece of the puzzle,” Frey said. “There’s a lot more work that needs to be done to research this aspect. A lot of that research is going to rely on the passage of time.”
ST. LOUIS — Researchers here have discovered cells in the bone marrow of COVID-19 survivors that were still producing antibodies against the virus months after recovery, suggesting immunity may last far longer than once thought.
The vanishing antibody mystery news has been invented by the FM/R/J Big Pharma mafia to promote vaccines as broadly as possible. (The SWISS R/J/FM mafia originally did vaccinate 2x all CoV-19 recovered...) We know from papers that people with SARS1 infections had no problem with with SARS2 (CoV-19) so corona immunity last up to 20 years. Also a Japanese study did confirm that CoV-19 recovered people - after 1 year - are at least 97% protected - far better than any vaccine.
Just to remind you: RNA vaccines after 1 shot give you "0" protection against the worst mutations and also after 2 shots they will not protect you from CoV-19 (from BRA/RSA/J mutation) only from a severe outcome.
Is heart inflammation a side effect of COVID-19 vaccine for adolescents? Utah doctor says it’s still unclear
https://www.deseret.com/platfo…or-says-its-still-unclear
While recent reports that inflammation of the heart muscle may be a side effect of the COVID-19 vaccine in some adolescents and young adults around the country are being reviewed by the Centers for Disease Control and Prevention, it’s business as usual for Utah’s vaccination program.
I haven’t heard any concern from the public regarding this,” Rich Lakin, Utah Department of Health immunization director, told the Deseret News Monday about the condition, known as myocarditis, reportedly occurring post-vaccination.
“We’re vaccinating more 12- to 15-year-olds than any other state. So the demand is there. Last week, it was at about 4% for the country, we were at around 9% of that age group,” Lakin said. Utah, which has the nation’s youngest population, has now fully vaccinated 45.3% of residents 12 and older.
The news about the possible adverse effect doesn’t mean parents should now wait to vaccinate their children, Lakin said. “They should continue vaccination. There hasn’t been anything from CDC that says to stop vaccinating. They’re looking into it. They don’t know if this is caused by the vaccination.”
The CDC’s vaccine safety advisory committee said May 17 there have been reports of myocarditis occurring after vaccination, predominantly in adolescents and young adults. No specifics were given, but the number of cases to date were described as “relatively few.”
Most cases appear to be mild, according to the committee, and more often follow the second and final dose of the vaccine, typically within four days, and are affecting more males than females. The rates of myocarditis reports after receiving the COVID-19 vaccination, the committee said, have not differed from the expected baseline rate.
“What you’re watching is the safety system in action. At the beginning, we don’t know what we’re really seeing,” said Dr. Andrew Pavia, chief of pediatric infectious diseases at University of Utah Health and director of epidemiology at Intermountain Primary Children’s Hospital. “We need to keep an open mind until we know more.”
His advice for parents?
“The clear message is that we don’t know if this is a real side effect yet. If it is, it’s going to be very, very rare. The risks of COVID for young people are still much greater than side effect we even can speculate seeing from the vaccine so far.”
Pavia said that if his adult children were still teenagers “and had an appointment tomorrow to get the vaccine, this would not give me any pause at all.”
Myocarditis, Pavia said, is usually seen in the spring and summer, with two or three cases a month expected at Primary Children’s Hospital. He declined to comment on whether any of the post-vaccination cases reported to the CDC occurred in Utah.
He said the cause commonly is a virus that circulates in the warmer months, but can also result from many other sources, including a reaction to the smallpox vaccine. Someone with myocarditis can experience chest pain, shortness of breath, coughs and fever, and is likely to recover without treatment.
Utahns 24 and younger account for more than a third of the state’s nearly 405,000 coronavirus cases, but less than 17 of the 2,292 lives lost to the virus. Health experts have said vaccinating younger residents is key to controlling the deadly disease.
“They help slow the spread. When they’re vaccinated, it helps protect the elderly. It helps protect the immunocompromised,” who can still be vulnerable to the virus even after being vaccinated themselves, Lakin said. “These kids play a big role in the way it spreads.”
Only the two-dose Pfizer vaccine is approved for use in the United States for those 16 and younger. In Utah, all residents at least 16 years old have been eligible to be vaccinated since mid-March, and shots to those as young as 12 became available earlier this month following CDC approval.
Lakin said shots are increasingly available from family doctors and pediatricians, as well as other long-standing locations including pharmacies, local health departments and mass vaccination sites, many of which are already winding down operations. He said adolescents and young adults are getting vaccinated at all of them.
Novel coronavirus really is seasonal, study suggests
By Rachael Rettner - Senior Writer 25 days ago
Warm temperatures and long hours of sunlight may reduce the spread of COVID-19, the study found.
https://www.livescience.com/cl…d-19-seasonal-spread.html
Warm temperatures and tropical climates may really help reduce the spread of COVID-9, a new study suggests.
The study found that places with warm temperatures and long hours of sunlight — such as countries close to the equator and those experiencing summer — had a lower rate of COVID-19 cases, compared with countries farther away from the equator and those experiencing colder weather.
The findings held even after the researchers took into account other factors that could affect both the spread of COVID-19 and the number of reported cases, such as a country's level of urbanization and the intensity of COVID-19 testing.
Still, the authors stress that their findings don't mean that summer weather will eliminate COVID-19; but it may give people a leg up against the disease.
"Our results do not imply that the disease will vanish during summer or will not affect countries close to the equator," the authors wrote in their paper, published April 27 in the journal Scientific Reports. "Rather, the higher temperatures and more intense UV [ultraviolet] radiation in summer are likely to support public health measures to contain SARS-CoV-2," the novel coronavirus causing COVID-19.
Seasonal virus
Shortly after the COVID-19 pandemic began in the winter of 2020, there was speculation that summer temperatures may bring relief from COVID-19. Indeed, many respiratory viruses, including flu viruses, show a seasonal pattern, peaking during the winter and dipping during the summer.
Scientists don't know for sure why these viruses follow a seasonal pattern, but a number of factors are thought to play a role. For example, studies suggest that many respiratory viruses are more stable and linger in the air longer in environments with cold temperatures and low humidity, Live Science previously reported. Human behaviors, such as gathering indoors in wintertime, could also boost transmission
Warm temperatures and tropical climates may really help reduce the spread of COVID-9, a new study suggests.
The study found that places with warm temperatures and long hours of sunlight — such as countries close to the equator and those experiencing summer — had a lower rate of COVID-19 cases, compared with countries farther away from the equator and those experiencing colder weather.
The findings held even after the researchers took into account other factors that could affect both the spread of COVID-19 and the number of reported cases, such as a country's level of urbanization and the intensity of COVID-19 testing.
Still, the authors stress that their findings don't mean that summer weather will eliminate COVID-19; but it may give people a leg up against the disease.
"Our results do not imply that the disease will vanish during summer or will not affect countries close to the equator," the authors wrote in their paper, published April 27 in the journal Scientific Reports. "Rather, the higher temperatures and more intense UV [ultraviolet] radiation in summer are likely to support public health measures to contain SARS-CoV-2," the novel coronavirus causing COVID-19.
Seasonal virus
Shortly after the COVID-19 pandemic began in the winter of 2020, there was speculation that summer temperatures may bring relief from COVID-19. Indeed, many respiratory viruses, including flu viruses, show a seasonal pattern, peaking during the winter and dipping during the summer.
Scientists don't know for sure why these viruses follow a seasonal pattern, but a number of factors are thought to play a role. For example, studies suggest that many respiratory viruses are more stable and linger in the air longer in environments with cold temperatures and low humidity, Live Science previously reported. Human behaviors, such as gathering indoors in wintertime, could also boost transmission
Studies in lab dishes have also found that high temperature and humidity reduce the survival of SARS-CoV-2, but whether this translates to real-world transmission was unclear.
In the new study, the researchers analyzed information from 117 countries, using data on the spread of COVID-19 from the beginning of the pandemic to Jan. 9, 2021. They used statistical methods to examine the relationship between a country's latitude — which affects the amount of sunlight it receives as well as temperature and humidity — and its level of COVID-19 spread. They also used data from the World Health Organization to control for factors that could affect how hard a country is hit by COVID-19, such as air travel, health care expenditure, the ratio of older adults to younger people and economic development.
They found that every 1 degree increase in a country's latitude from the equator was tied to a 4.3% increase in the number of COVID-19 cases per million people. This means that if one country is 620 miles (1,000 kilometers) closer to the equator compared with another, the country closer to the equator could expect to have 33% fewer COVID-19 cases per million people, with all other factors being equal between the countries.
