So one has to wonder if, according to the WHO, 1902 children under the age of 5 have died due to Covid worldwide (likely most with severe co-morbidities) and if about 1,000 kids have come down with hepatitis (and that is just one of many other harms) that have been caused by lockdowns, was it worth it?
Too bad it wasn't established if the hepatitis outbreak locations correlate well to places of relatively severe lockdown measures for children.
The original article is here :
Conclusions/final statements
AAV2 detected independently by sequencing and by real-time PCR was present at significantly higher frequency in cases of paediatric non-A-E hepatitis than controls. That this occurs in the context of an increased frequency of the DRB1*04:01 allele when compared to a local blood donor control population requires further investigation, but points to a T helper cell-mediated pathological response triggered by exposure to HAdV and/or AAV2 infection.
AAV2 is a small non-enveloped single-stranded DNA virus of 4,675 nucleotides belonging to the species adeno-associated dependoparvovirus A (genus Dependoparvovirus, family Parvoviridae)11, first described in 1965 and occurs in up to 80% of the adult population, seroconversion occurring in early childhood following respiratory infection12
Btw, AAV2 has been used as a viral vector in the past for gene delivery.
I'm surprised no mention was made in the paper of the adenovirus vector for the Astra Zeneca vaccine. It's a modified cold virus of chimpanzees and which is novel to humans. But humans will make antibodies to it, and who knows, these antibodies might be triggering some kind of auto-immune condition against the liver. I know, children didn't receive this vaccine, but there's no guarantee it didn't gain some functionality by recombination and become transmissible to some degree. I say this because the UK has been the hotspot for the hepatitis, and also the the Astra Zeneca vaccine.