GROWING EVIDENCE OF MYOCARDITIS RISK IN YOUNG PEOPLE AND ATHLETES

GROWING EVIDENCE OF MYOCARDITIS RISK IN YOUNG PEOPLE AND ATHLETES

A new trend in heart disease is emerging in younger people, particularly athletes, thought to be a result of mRNA COVID-19 vaccination. The South Korean

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A new trend in heart disease is emerging in younger people, particularly athletes, thought to be a result of mRNA COVID-19 vaccination. The South Korean Ministry of Food and Drug Safety reported 492 deaths in young people directly after vaccination, which TrialSite reported on in September. In the same month, TrialSite reported on a report from Public Health Ontario (PHO) on aggregating COVID-19 vaccination adverse events following vaccination (AEFI) across Ontario, with young persons aged 24 and under accounting for 80% of the total AEFIs. Now, further reports are emerging on cardiac events in young vaccinated people around the world. Are the reports accurate? If does this change the risk-benefit analyses used for aggressive universal vaccination programs?

The German newspaper Berliner Zeitung has published an investigation into why numerous professional and amateur soccer players have recently collapsed. The report noted 24 recent incidents of footballers who had cardiac problems or collapsed on the field, some of which responded to cardiopulmonary resuscitation, and others resulting in death. Cardiac arrest was shown to be the most common cause. As well as the soccer players on the field, non-players such as coaches and referees counted for eight of the occurrences.

The Covid World, a website that gives a voice to victims and survivors of adverse effects of COVID-19 vaccines, lists eleven world-class athletes who died or were severely injured after receiving the COVID-19 vaccine. A baseball player from Japan and an archery athlete from Malaysia died in separate situations, weeks after receiving COVID-19 vaccinations. The majority of the instances include sportsmen who suffer cardiac problems after receiving the COVID-19 vaccination, such as pericarditis and myocarditis. In another four cases, blood clots and other health issues occurred.

Peter Schirmacher of the University of Heidelberg in Germany argues that total vaccination-related deaths are underreported. He believes that the vaccination is responsible for 30-40% of the 40 autopsies of persons who died within two weeks of receiving a COVID-19 vaccination in his research, as previously reported on TrialSite. COVID vaccinations that are based on genes induce the body to create spike protein, which is toxic and can cause inflammation and blood clots in all major organs, including the brain, heart, lungs, and ovaries, for up to 48 hours. In the same research, 42% of the 400,000 adverse events reported to the Vaccine Adverse Event Reporting System (VAERS) in relation to COVID-19 vaccinations had at least one cardiovascular incident.

A Link Between Heart Problems and mRNA Vaccine

For a while the World Health Organization (WHO) failed to emphasize heart issues as an adverse event of special interest for mRNA vaccines, however the global health agency updated its position on October 27 highlighting the “rare” but relevant topic. WHO previously identified anaphylaxis in association with mRNA vaccines. The AstraZeneca and Janssen COVID-19 adenovirus vector vaccines have been linked to a highly rare and atypical clotting syndrome with thromboembolic events (blood clots) and thrombocytopenia (low blood platelet count). Thrombosis with Thrombocytopenia Syndrome is the name given to this illness (TTS).

However, the Advisory Committee on Immunization Practices of the Centers for Disease control (CDC), found a “likely association” between the Pfizer and Moderna vaccines and reported cases of heart inflammation. Cases of myocarditis have been reported to the VAERS following mRNA COVID-19 vaccination (Pfizer-BioNTech or Moderna), particularly in male teenagers and young adults, more often after the second dose and usually within a few days of vaccination.

Between January and April 2021, patients in the US Military Health System who developed myocarditis after receiving COVID-19 vaccine were investigated in a case series. Following receipt of an mRNA COVID-19 vaccination, myocarditis developed in previously healthy military patients with identical clinical symptoms. After a second vaccine injection, the number of male military members with myocarditis cases was higher than projected.

However, more monitoring and study of this adverse occurrence after vaccination is required. Another study linked the vaccination to an increased risk of myocarditis, with 1 to 5 events per 100,000 persons. After SARS-CoV-2 infection, the risk of this potentially serious adverse event, as well as many other serious adverse events, was significantly raised.

Heart Disease Trend Emerging in Younger People

According to statistics reported by researchers at the Centers for Disease Control and Prevention, the coronavirus vaccinations developed by Pfizer-BioNTech and Moderna may have caused heart problems in over 1,200 Americans, including over 500 under the age of 30.

Some young individuals who have had COVID-19, those who have been vaccinated against the virus, and student athletes are showing signs of cardiac disease, according to health professionals. In a joint statement released by the International Coalition of Medicines Regulatory Authorities and WHO, incidences of thromboembolic events with thrombocytopenia following vaccination were mostly recorded in younger people rather than the elderly. In the same source stated that even public health officials in several countries advise against administering the AstraZeneca vaccine to younger individuals.

When reviewing VAERS data, there are several limitations. For starters, anyone can contribute information willingly, therefore reports range in quality and thoroughness. Second, because VAERS only receives reports for a small fraction of real adverse events, one of the key possibilities is underreporting. Finally, VAERS accepts all reports without deciding if the vaccine was to blame. VAERS will accept the report without requiring confirmation that the occurrence was caused by the vaccination.

Impact of Vaccine on Athletic Performance

Vaccinated patients who have never been exposed to SARS-CoV-2 may have elevated physiological demands for at least 2 to 3 weeks after receiving their second dose of the mRNA vaccine. In the post-vaccine study that included 18 healthy adults (nine females and nine men) ranging in age from 24 to 43 years old, oxygen uptake, CO2 production, respiratory exchange ratio, ventilation, heart rate, serum noradrenaline, and rating of perceived exertion were all considerably higher. After vaccination, exercise adrenaline levels were considerably lower, and serum lactate levels were trending lower, suggesting that the body was not adapting well to exercise conditions.

Unfortunately, because the total number of cases of COVID-19 is unknown, it is difficult to estimate how likely someone is to die if they become infected. The fact that not everyone with COVID-19 is tested is one of the major reasons behind this. However, vaccine-related myocarditis was found in 1.0 per 100 000 people who received at least one COVID-19 vaccination, while pericarditis was found in 1.8 per 100 000 people who received at least one COVID-19 vaccination.

‘Still Safe’, say the Authorities

Despite these data, health authorities continue to recommend vaccinations. In September, TrialSite reported that the South Korean government had only acknowledged two post-vaccination deaths as being related to the vaccines; other governments have followed suit, quickly assuring the public that the deaths are not linked to the COVID-19 vaccine.

The American Medical Society for Sports Medicine (AMSSM) assembled an expert panel to discuss the current data, knowledge gaps, and recommendations around COVID vaccination in athletes. They concluded that COVID vaccination should be included during the pre-participation physical examination for athletes at all levels of training and competition, according to a document released in November 2021.

Although there should be caution in the event of rare side effects such as myocarditis following COVID-19 vaccination, the CDC continues to recommend that everyone aged 12 and above get vaccinated against COVID-19, on the basis of benefits outweighing the risks of a rare adverse reaction to vaccination, such as myocarditis or pericarditis

German Newspaper Highlights “Unusually Large” Number of Soccer Players Who Have Collapsed Recently - [your]NEWS

Publishes huge list of footballers who have recently collapsed or died.

yournews.com

A List of World Class Athletes Who Died Or Suffered Severe Injuries After COVID-19 Vaccine

A list of top athletes from around the world that, in just the last few weeks and months, have either died or been left injured by the COVID-19 vaccines.

thecovidworld.com

In Germany, Some Groups Suspect COVID-19 Vaccination Deaths are Undercounted but Not the Majority

Peter Schirmacher with the University of Heidelberg in Germany reports that his group has studied 40 people who have died within two weeks of vaccination;

trialsitenews.com

The Link Between Myocarditis and COVID-19 mRNA Vaccines

As cases of myocarditis are being monitored, the benefits of mRNA COVID-19 vaccination far outweigh the risk of myocarditis, doctors say.

www.yalemedicine.org

Why not add some reports on myocarditis caused by Covid-19 infection? Just to balance this a bit...(although I expect this to be called CDC propaganda... nad it may not really comparable due to missing vaccinations)

"During March 2020–January 2021, patients with COVID-19 had nearly 16 times the risk for myocarditis compared with patients who did not have COVID-19, and risk varied by sex and age."

Association Between COVID-19 and Myocarditis...

Viral infections are a common cause of myocarditis. Some studies have indicated an association between COVID-19 and myocarditis.

www.cdc.gov

Conclusions
Myocarditis (or pericarditis or myopericarditis) from primary COVID19 infection occurred at a rate as high as 450 per million in young males. Young males infected with the virus are up 6 times more likely to develop myocarditis as those who have received the vaccine.

A plant based chewing gum? Beginning to think chewing on leaves and twigs offer more protection than our latest and greatest vaccines!!!!!!!

University of Pennsylvania-led Study Team Reveal ACE2 Chewing Gum Just Might Inhibit SARS-CoV-2 Oral Transmission

University of Pennsylvania-led Study Team Reveal ACE2 Chewing Gum Just Might Inhibit SARS-CoV-2 Oral Transmission

A study led by the University of Pennsylvania, Department of Medicine, and other research institutes such as Wistar Institute sought to advance a chewing

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A study led by the University of Pennsylvania, Department of Medicine, and other research institutes such as Wistar Institute sought to advance a chewing gum that could decrease oral transmission and infection associated with COVID-19. A prominent group of esteemed scientists and physicians suggest that chewing gum with virus-tapping proteins could introduce an economical approach to protecting patients from a majority of oral-based viral reinfections via “debulking” or minimizing transmission to others.

TrialSite briefly breaks down this research for all.

What is the issue?

The SARS-CoV-2 virus spreads through droplets and/or aerosol transmission and is most concerning in close quarters where people are close together. Infected individuals more easily spread it to others or “transmit” the pathogen infecting others.

What attempts have been made to inhibit this problem thus far?

Masking and physician distancing has been the primary means that public health authorities have sought to reduce the spread of COVID-19. However, some projects in public setting buildings involve the improvement of air exchanges via filtration.

What is the primary way the pathogen is spread?

The most contagious scenario is in closed quarters where people talk, breathe, or cough near someone else. In fact, according to the recent study, most people emit >100 times smaller aerosols (< μm) during these interactions.

What about vaccination?

Well, most of the world isn’t’ vaccinated, and frankly, it was never reasonable that the World Health Organization and other pandemic preparation and response teams to think that 70%+ of the world could be vaccinated within a year or two—it is just unreasonable and hints of removed elites that don’t understand how the world works.

In fact, vaccine hesitancy continues to be a big problem, especially in cultures where Western medicine isn’t trusted. Moreover, Western governments have made profound mistakes in positioning the vaccine as a cure. It is not—rather, it could be a useful measure, such as an influenza shot on an annual basis.

But importantly, the influenza shot doesn’t stop all people from becoming infected with the flu. Just like the current crop of COVID-19 vaccines don’t work too well after several months. Vaccine effectiveness wanes, and boosters are required. Yet, what are the long-term consequences of this regimen? We do not know yet.

Vaccination is an important strategy to reduce hospitalization and death, but it doesn’t stop viral transmission, especially after several months.

What is causing viral strain mutation?

There are different causes. First, the coronavirus pathogen itself is known to mutate—that’s a given. But some believe that the lack of universal vaccination creates reservoirs for mutation. Yet that mutation would happen anyway! Other scientists such as Geert Vanden Bossche believe that the act of mass vaccination in the pandemic itself triggers the conditions for further mutation. There are no studies that prove one argument over the other conclusively.

What is the pressing need?

The reality is that a highly contagious virus has caused over 5 million deaths and 200 million or more infections, thus the need for a way to develop measures to slow down the transmission of infection associated with SARS-CoV-2.

Why did the researchers in this study focus on the oral cavity?

Because that is where the primary site of viral replication occurs.

What did the researchers do?

They sought to “advance a novel concept of debulking virus in the oral activity with virus-tapping proteins CTB-ACE2 expressed in chloroplasts to develop clinical-grade plant material to meet FDA requirements.

What is the investigational product?

Chewing gum (2 g) containing plant cells expressed CTB-ACE2 up to 17 mg ACE/g dry weight (11.7% leaf protein). This investigational product has physical characteristics as well as the taste and flavor like standard chewing gum products. The authors report “no protein was lost during gum compression.”

What were the results of this study?

According to the authors, CTB-ACE2 gum efficiency (>95%) inhibited entry of lentivirus spike or VSV spike pseudovirus in Vero/CHO cells when quantified by luciferase or red fluorescence.

Moreover, they report, “Incubation of CTB-ACE2 microparticles reduced SARS-CoV-2 virus count in COVID-19 swab/saliva samples by >95% when evaluated by microbubbles (femtomolar concentration) or qPCR, demonstrating both virus trapping and blocking of cellular entry.”

Additionally, the authors pointed out that when compared with healthy individuals, COVID-19 saliva samples demonstrated “low or undetectable ACE2 activity (2,582 versus 50,126 ΔRFU; 27 versus 225 enzyme units),” verifying “greater susceptibility of infected patients for viral entry.”

Finally, they note “CTB-ACE2 activity was completely inhibited by pre-incubation with SARS-CoV-2 receptor binding domain, offering an explanation for reduced saliva ACE2 activity among COVID-19 patients.”

What is the authors’ key takeaway?

Chewing gum with “virus-tapping proteins” may represent an economical approach to protect patients from most oral virus-reinfections via “debulking or minimizing transmission” to other people nearby.

Who funded the study?

∙ Research in the Daniell laboratory is supported by funding from NIH grants R01 HL 107904 , R01 HL 109442 , and R01 HL 133191

∙ Commonwealth of Pennsylvania, Department of Community and Economic Development grant one corresponding author Henry Daniell, W.D. Miller Professor, Vice-Chair, Department of Basic and Translational Services, University of Pennsylvania on “COVID-19 Pennsylvania Discoveries: Responding to SARS-CoV-2 Through Innovation & Commercialization” funded the purchase of freeze dryers, toxicology studies on ACE2 produced at Fraunhofer USA/AeroFarms, and production of the chewing gum.

∙ Grant supported saliva sample collection in the Collman Lab

∙ Research in the Harty laboratory is supported by funding from a University of Pennsylvania School of Veterinary Medicine COVID-19 Pilot Award, a Mercatus Center award, and NIH T32 grant AI070077 to Ariel Shepley-McTaggart.

∙ Pen Center for Precision Medicine, Penn Health-Tech, Penn Center for Innovation and Precision Dentistry and NIH RADx program funding supported research in the Wang laboratory.

Lead Research/Investigator (Corresponding Author)

Henry Daniel, PhD, W.D. Miller Professor, Vice-Chair, Department of Basic and Translational Services, University of Pennsylvania, Dental Medicine

Debulking SARS-CoV-2 in saliva using angiotensin converting enzyme 2 in chewing gum to decrease oral virus transmission and infection

DEFINE_ME

Quote from zorud

Why not add some reports on myocarditis caused by Covid-19 infection? Just to balance this a bit...(although I expect to this to be called CDC propaganda....)

"During March 2020–January 2021, patients with COVID-19 had nearly 16 times the risk for myocarditis compared with patients who did not have COVID-19, and risk varied by sex and age."

https://www.cdc.gov/mmwr/volumes/70/wr/mm7035e5.htm

No one denies this but it's not the point. Vaccinating healthy people can lead to myocarditis and longer term problems. So I guess you take the Huxley/rothwell approach that these people are just expected stats. It's one thing for the virus to cause problems but for the cure to cause the same problems? That is very concerning!

French Research and Real-World Data Counter Omicron Hysteria

French Research and Real-World Data Counter Omicron Hysteria

Joel S. Hirschhorn So much talk about Omicron; so much fear mongering; so much talk about science. Most is nonsense. The best research has received

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So much talk about Omicron; so much fear mongering; so much talk about science. Most is nonsense. The best research has received little attention. It comes from esteemed, senior French scientist: Dr. Jacques Fantini, Professor of Biochemistry and Molecular Biology at the University of Aix-Marseille.

You are about to learn what senior people in the public health establishment need to use, especially Fauci, who claims he speaks for and represents “science.” If he knows the French research, he is not sharing it with the public, nor is the mainstream media.

The key scientific achievement by Fantini is the calculation of one key parameter he calls the index of transmissibility (T) of a COVID variant. The key work was published in June 2021 with the title “Structural dynamics of SARS-CoV-2 variants: A health monitoring strategy for anticipating Covid-19 outbreaks.” This research is very sophisticated, detailed, and challenging. Genomic sequence data are used in the analyses of variants.

The molecular details of variants are analyzed to calculate T values for COVID variants. Originally, T values for known variants or strains of the COVID virus were determined. The T value for the Delta variant done in early 2021 accurately predicted the surge of Delta throughout the world, making it the dominant variant in many countries, including the US.

The T value accurately describes to what extent a variant is or is not very transmissible. The higher the value of T, the greater is the ease at which a variant is spread from one person to another. The higher the value, the more contagious is the variant. Fantini said how T values could serve a critical need: “T-index can be used as a health monitoring strategy to anticipate future Covid-19 outbreaks.” At this moment, the question is “Is the T value for Omicron of concern?”

So, now look at the following table that gives T values for the original five variants published by Fantini, plus what he has just released for the new Omicron variant.

Variant T-index

Initial Wuhan 2.16

UK – Alpha 3.59

Brazil – Gamma 3.65

South Africa – Beta 3.82

Delta 10.67

Omicron 3.90

Delta stands out for having an extremely high T value compared to previous variants. No surprise that it quickly became the dominant variant globally.

And equally impressive is the relatively low T value for Omicron, just 37% of the Delta value. Omicron should not be of high concern by people and nations. It is in line with most pre-Delta variants. It is not exceptional. There is no scientific basis for all the hysteria about Omicron. As shown below, most people assessed with Omicron were vaccinated and got breakthrough infections showing vaccines offer little protection.

Additional observations

Note that the higher the T value it is also likely the less effective are current vaccines for defending against the variant and protecting people from it (as real-world data given below show). However, the higher T value does not imply greater lethality. As is known by virologists, variants are smart enough to not kill their victims, which would also kill them and prevent them from spreading. Thus, high T value variants spread easily, can cause health impacts, but do not necessarily kill people at a high rate.

Fantini said this: “For Omicron, the mutations go in all directions, without any particular logic, some annihilating each other. The mutational profiles …suggest that neutralizing antibodies [from vaccine immunity] will have very low activity on this variant. This analysis of the Omicron variant suggests that this variant will not supplant Delta.” In other words, with far less spreading potential, Omicron is not likely to replace the much higher transmissible Delta prevalent globally. Even though reports keep coming in from different nations that Omicron has been found.

More positive insights had to do with the more than 30 mutations and exactly where they were in the molecule. “The affinity of the Omicron … for ACE-2 [cellular material that causes infection] is decreased compared to all other variants analyzed to date, probably as a consequence of this accumulation of mutations.” Fantini is saying that Omicron is not only not as highly transmissible as Delta, it is also not as infectious.

Worth remembering is that all the current COVID vaccines were designed to address the earliest COVID virus molecule. Thus, they do not protect very well against later variants that have considerable mutations. Is protection zero? No. Current vaccines offer limited defense against variants because they only aim at a small fraction of the virus molecule components.

Vaccine problems

In a more recent article, Fantini and an associate said: there is a “progressive loss of immunity induced by the two doses of vaccines directed against the spike protein” because current vaccines are not designed to defend against recent variants, including Delta and Omicron. Moreover, “the third vaccine [booster] dose can have serious long-term side effects due to the “ADE” phenomenon (Antibody-dependent enhancement: facilitation of infection by antibodies). The benefit/risk ratio would be unfavorable.” In other words, like other researchers, they see the negative impact of current COVID vaccines that reduce protection offered by a person’s immune system. What is being said is that antibodies not only offer little protection but, instead, facilitate viral infection and promote release of new mutations or variants. This is consistent with considerable data showing correlations between higher vaccination rates and higher death rates at the nation level.

This too was noted: “The immune response to SARS-CoV-2, whether natural or vaccine-induced, produces antibodies directed against the spike protein. In the case of mRNA vaccines, the only molecular target is the spike protein. In the case of natural infection with the virus, the immune response [natural immunity] is directed against several viral proteins, including the spike protein. In all cases, the spike protein is therefore crucial. However, SARS-CoV-2 is an RNA virus that mutates a lot, and many mutations affect the spike protein, which disturbs its recognition by antibodies.” The bottom line is that vaccine immunity is inferior to natural immunity, because the former was designed for the earliest strain and only targets a small fraction of the complex COVID molecule.

Real world data show no severe illness and no protection from vaccines

The forecast by Fantini about Omicron is consistent with information flowing in. Specifically, vaccines will have little impact on Omicron transmission or infectivity. For example, Reuters reported: “Four people in southern Germany have tested positive for the Omicron COVID-19 variant even though they were fully vaccinated against the coronavirus said officials.” Moreover, “All four showed moderate symptoms.”

Previously, it was highlighted, according to the Botswana government, the Omicron variant was first detected in four people who were fully vaccinated. Information from South Africa is that Omicron caused mild symptoms and no patients needed hospitalization, and that the European Union’s public health body said that they’ve found 44 cases containing the omicron variant in 10 of their member countries, all of which had mild or asymptomatic illness.”

Also reported was that “Two quarantined travelers in Hong Kong who have tested positive for the variant were vaccinated with the Pfizer jab. All three initial confirmed and suspected cases reported from Israel occurred among fully vaccinated individuals. An Israeli doctor revealed that he had been infected with Omicron despite being triple vaccinated also wearing a mask.

In Australia, “New South Wales state authorities reported that two travelers from South Africa to Sydney had become Australia’s first omicron cases. Both were fully vaccinated, showed no symptoms.” A person in San Francisco was reported to have traveled from South Africa, had mild symptoms and had been vaccinated. Interestingly, officials said they had contacted everyone who had close contact with the person, and they had all tested negative.

Meanwhile, everything that Fauci has said is completely inconsistent with actual data as well as what Fantini has forecast. Everything he has said seems clearly aimed at instilling fear about Omicron so that invasive, authoritarian government actions and continued push for vaccines could be justified.

Conclusions

A review of studies found unequivocally that COVID vaccines do not stop viral transmission, with no difference between vaccinated and unvaccinated people. So, all real-world evidence is that Omicron cannot be effectively addressed by COVID vaccines. Together with Fantini’s work the proper conclusion it that Omicron will not be very transmissible nor be more infective than Delta.

Because mutations will continue to produce variants, it is critically important to use the work of Fantini to accurately assess whether or not a new variant should evoke the fears and government responses that have sprung up so quickly for Omicron.

Dr. Joel S. Hirschhorn, author of Pandemic Blunder and many articles and podcasts on the pandemic, worked on health issues for decades, and his Pandemic Blunder Newsletter is on Substack. As a full professor at the University of Wisconsin, Madison, he directed a medical research program between the colleges of engineering and medicine. As a senior official at the Congressional Office of Technology Assessment and the National Governors Association, he directed major studies on health-related subjects; he testified at over 50 US Senate and House hearings and authored hundreds of articles and op-ed articles in major newspapers. He has served as an executive volunteer at a major hospital for more than 10 years. He is a member of the Association of American Physicians and Surgeons, and America’s Frontline Doctors

Quote from zorud

It seems there is some exceptional, better to say exponential.... 300% rise of cases

Today's news in Switzerland. So far - in RSA - no hospitalization ==> death from Omicron,.. but very young children are more sensitive. More - 100 so far - did end up in Hospital. Omicron + RSV? Same picture as classic old corona where the youngest also end up in hospital.

Let's hope that we now have the long living endemic virus and that it spreads fast!!!

Also Swiss news:: We now have the pandemic of the vaccinated. Final confession...

Quote from zorud

Myocarditis (or pericarditis or myopericarditis) from primary COVID19 infection occurred at a rate as high as 450 per million in young males.

This figure 450 definitely is not given by CDC.

citation:

Among the 2,116 patients with COVID-19 and myocarditis, 1,895 (89.6%) received a diagnosis of COVID-19 and myocarditis during the same month; the remaining patients received a myocarditis diagnosis 1 month (139; 6.6%) or ≥2 months (82; 3.9%) after their COVID-19 diagnosis.

The findings in this study are subject to at least six limitations. First, the risk estimates from this study reflect the risk for myocarditis among persons who received a diagnosis of COVID-19 during an outpatient or inpatient health care encounter and do not reflect the risk among all persons who had COVID-19. Second, misclassification of COVID-19 and myocarditis is possible because conditions were determined by ICD-10-CM codes, which were not confirmed by clinical data (e.g., laboratory tests or cardiac imaging) and could be improperly coded or coded with a related condition (e.g., pericarditis). Third, encounters for COVID-19, myocarditis, and COVID-19 vaccination occurring outside of hospital systems that contribute to PHD-SR are not included within this data set. Fourth, underlying medical conditions and alternative etiologies for myocarditis (e.g., autoimmune disease) were not ascertained or excluded. Fifth, the obtained measures of association could be biased because of the choice of the comparison group (all patients without COVID-19) and if physicians were more likely to suspect or diagnose myocarditis among patients with COVID-19.

> 50% of the cases are in patients. Because all hospitals use prednisone we expect far more cases of other viral infections. Prednisone is a very strong immune suppressive. The study does also not properly track the vaccine status - the biggest mistake - just look up database. Same months only after CoV-19 is relevant.

To make it clear. A strong viral infection always potentially will lead to hart inflammation. But the viral infections did obviously not lead to death!

The spike protein shed by all attacked cells - over months - directly attacks the hearts endothelian cells. This attack by a far higher number very often leads to death. In the CDC study also age 25..35 is the least affected, where in vaccination its the strongest (death) affected.

Korea definitely has to stop vaccinate age < 40! There seem to be some genetic issues. Also seen in Bangladesh in relation with CoV-19.

Only treatment - Ivermectin - will help!

Quote from Wyttenbach

Today's news in Switzerland. So far - in RSA - no hospitalization ==> death from Omicron,.. but very young children are more sensitive. More - 100 so far - did end up in Hospital. Omicron + RSV? Same picture as classic old corona where the youngest also end up in hospital.

Let's hope that we now have the long living endemic virus and that it spreads fast!!!

Also Swiss news:: We now have the pandemic of the vaccinated. Final confession...

???

Quote from zorud

???

I just repeated todays news ::

Es ist wichtig, dass wir jetzt mit den Boostern vorwärtsmachen. Autor:

Es wird langsam eine Epidemie der Geimpften, die sich in den ersten Monaten dieses Jahres haben impfen lassen. Deshalb ist es wichtig, dass wir jetzt mit den Boostern vorwärtsmachen.

Infektiologe im Interview - Genügen die neuen Corona-Massnahmen des Bundes, Herr Cerny?

Heute wird der Bundesrat die Corona-Massnahmen verschärfen. «Es wurde höchste Zeit», sagt der Virologe Andreas Cerny.

www.srf.ch

Further unknown (= documents not ready) has to be added ( at least 80%) to vaccinated. So its about 50:50 in cases with about 68% vaccination over all an 80% in the relevant population. But at least 50% are recovered... So vaccine protection is the same as everywhere except Netherlands that claims a fantastic protection we see nowhere else...

You did not repeat the news...you interpreted the news, didn't you?

("We now have ...." and "es wird langsam..." is quite a difference.... )

Quote from zorud

Conclusions
Myocarditis (or pericarditis or myopericarditis) from primary COVID19 infection occurred at a rate as high as 450 per million in young males. Young males infected with the virus are up 6 times more likely to develop myocarditis as those who have received the vaccine.

The false equivalence in the article is painful to behold. Myocarditis from a viral infection is very different than myocarditis from the vaccine.

Dr. Peter McCullough: Myocarditis From Vaccines Is Nothing Like What We See From Natural Infection

Follow us on GAB: https://gab.com/redvoicemedia Follow Us On Telegram: https://t.me/redvoicemedia Real News & Commentary for Real Patriots:…

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“Dr. Peter McCullough: Now, the myocarditis that occurs with a natural infection is usually those sick enough to be in the ICU, and it’s a troponin elevation only. It is very different than the myocarditis that we’re seeing with the vaccines, which we’ll get to. The myocarditis in COVID-19 is mild, it’s inconsequential, and it’s largely a troponin elevation. I don’t want anybody to think that the myocarditis of the natural infection is anything like what we’re seeing with the vaccines.

Interviewer: Exactly. The vaccine produces the inflammatory type process is on the heart.

Dr. Peter McCullough: And the vaccine is directly there. Now there’s preclinical studies suggesting the lipid nanoparticles actually go right into the heart. The heart expresses the spike protein. The body attacks the heart. There are dramatic EKG changes. The troponin, the blood test for heart injury with the vaccine myocarditis, is 10 to 100 fold higher than the troponin we see with the natural infection. It’s a totally different syndrome. About what when the kids get myocarditis after the vaccine, 90% have to be hospitalized. They have dramatic EKG changes, chest pain, early heart failure. They need echocardiograms. If the ejection fraction is low, they need medications to prevent heart failure. So vaccine-induced myocarditis is a big deal. And in children, it’s way more serious and more prominent than a post-COVID myocarditis.”

Quote from Fm1

Bangladesh experienced a few spikes during the COVID-19 pandemic, including a particularly bad one during the summer of 2021. Still, they have turned it around in a similar way as the state of Uttar Pradesh in India.

Perhaps people in Bangladesh are often infected with parasites, but not diagnosed. I wouldn't know about that. Perhaps the population there is young, so not many people die. Or, perhaps it is like Uttar Pradesh in India insofar as the statistics are meaningless, and deaths are not even detected, or they are covered up.

I don't know why the numbers are low, but I am sure that ivermectin has no effect on COVID. That is what the double-blind tests show. You cannot argue with that. If there was a significant effect, it would be measurable.

Quote from Fm1

Vaccinating healthy people can lead to myocarditis and longer term problems.

The myocarditis is rare and there have been no serious cases. Patients likely to have it can be identified and monitored. They are mostly young men. There are no longer term problems.

Quote from JedRothwell

Perhaps people in Bangladesh are often infected with parasites, but not diagnosed. I wouldn't know about that. Perhaps the population there is young, so not many people die. Or, perhaps it is like Uttar Pradesh in India insofar as the statistics are meaningless, and deaths are not even detected, or they are covered up.

I don't know why the numbers are low, but I am sure that ivermectin has no effect on COVID. That is what the double-blind tests show. You cannot argue with that. If there was a significant effect, it would be measurable.

Oh please provide the one study that shows no effect and I'll show you the now 67 studies that do show a large effect. You remind me of cold fusion deniers!

Does Vitamin D Help Fight COVID-19 Severity? Istanbul University-Cerrahpasa Led Study Suggests So

Does Vitamin D Help Fight COVID-19 Severity? Istanbul University-Cerrahpasa Led Study Suggests So

A group of biochemists, endocrinologists, pharmacists, physicians, and other academic medical specialists from Istanbul University-Cerrahpasa in Istanbul,

trialsitenews.com

A group of biochemists, endocrinologists, pharmacists, physicians, and other academic medical specialists from Istanbul University-Cerrahpasa in Istanbul, Turkey recently pursued the medical research question of whether vitamin D supplementation could improve COVID-19 patient outcomes. The authors conducted a prospective, observational study involving a retrospective analysis of 867 healthy COVID-19 patients. From that data the Turkey-based team of investigators identified 23 healthy individuals as well as 210 cases, observing that 163 of the patients were administered vitamin D supplementation. 95 of these patients were monitored for 14 days with the study’s protocol delineating the following clinical outcomes: routine blood markers, serum levels of vitamin D metabolism, and action mechanism-related parameters. The research team found that vitamin D supplementation not only shorted hospital stay for the COVID-19 patients but also decreased mortality, even with those identified with comorbidities. Thus, given the study results, the team concluded that vitamin D is an effective treatment materially impacting the identified target clinical parameters. Vitamin D is an essential COVID-19 treatment, concluded the authors of the study published in peer-reviewed journal Nutrients.

This observational class of study, of course, doesn’t provide strong evidence as an appropriately statistically powered randomized controlled trial, but nonetheless, can provide valuable data points that help evince the benefits of a particular treatment. The study authors pursued an important question for the lay population of whether vitamin D is a positive supplement for addressing COVID-19.

A conflicting body of research involves the case for the vitamin that consists of a group of fat-soluble secosteroids involved with the boosting of intestinal absorption of calcium, magnesium, and phosphate in addition to numerous other biological results. In humans, vitamin D3 also known as cholecalciferol, and vitamin D22, known as ergocalciferol, are most vital according to information from the National Institutes of Health (NIH).

What about Vitamin D & COVID-19?

Of note, in 2017 an international team of scientists conducted a meta-analysis of 25 eligible randomized controlled trials involving 11,321 participants to determine if vitamin D supplementation reduced the risk of acute respiratory tract infection among all participants. Published in the peer-reviewed BMJ, the study team concluded that vitamin D was not only safe but also based on the data from 25 studies demonstrated effectiveness at protecting against acute respiratory tract infection overall.

Back in May of this year, TrialSite reviewed a Spanish study led by a team of scientists at the University of Cordoba that found significant changes in mortality for patients receiving calcifediol compared to patients not receiving the supplement.

Meanwhile, a total of 87 studies involving vitamin D and COVID-19 are disclosed in the clinical trials registry maintained by the U.S. National Library of Medicine (NIH). 29 of those studies have been completed, and TrialSite will update the community as more findings become available.

A number of investigations have inquired into the role of vitamin D deficiency as well as the prospects of the benefit of the use of this substance as a supplemental treatment during the COVID-19 pandemic. For example, numerous meta-analyses investigate the relationship between adverse outcomes in COVID-19 and vitamin D deficiency. Examples of these studies include studies out of Iran and Brazil. A couple of meta-analyses covering approximately 40 studies demonstrated the greater risks of COVID-19 infection in vitamin D deficient persons including (Kazemi, Mohammad) and (Petrelli et. Al.)

However, the NIH has declared that there is “insufficient evidence to recommend either for or against the use of vitamin D for the prevention or treatment of COVID-19.” Moreover, the UK National Institute for Health and Care Excellence (NICE) has a similar stance to the NIH.

The Istanbul University-Cerrahpasa Findings

In the Istanbul University-Cerrahpasa vitamin D study the authors reported in the peer-reviewed journal Nutrients that “the treatment protocol increased the serum 25OHD levels significantly to above 40 ng/mL within two weeks.” They reported that COVID-19 patients not treated with vitamin D faced a 1.9-fold increased risk of hospitalization over 8 days compared with those patients that received the vitamin D supplemental treatment.

The authors report that the vitamin D cohort benefited from a decreased mortality rate by 2.4 times. Because of the findings of specific serum biomarkers with 250HD, the authors propose that the study supplement impact on COVID-19 could possibly involve regulation of a series of relevant genes and proteins such as INOS1, IL1b, IFng, cathelicidin-LL37, and ICAM1.

About the Research Center

Based in Turkey’s largest city Istanbul, Istanbul University-Cerrahpasa was part of the vast university system called Istanbul University. Istanbul University was established in 1453. The Cerrahpasa campus is named after the head of the Ottoman empire from 1598 to 1599 as reported in Wikipedia.

Lead Research/Investigator

Corresponding authors included:

Spyridon N. Karras, MD, Ph.D., Endocrinology and Metabolism Unit, Department of Internal Medicine, Cerrahpasa Faculty of Medicine, Istanbul University-Cerrahpasa; Department of Infectious Diseases and Clinical Microbiology, Cerrahpasa Faculty of Medicine

Duygu Gezen-Ak, Ph.D., Brain and Neurodegenerative Disorders Research Laboratories, Department of Medical Biology, Cerrahpasa Faculty of Medicine

Erdinc Dursun, Ph.D., Brain and Neurodegenerative Disorders Research Laboratories, Department of Medical Biology, Cerrahpasa Faculty of Medicine, Istanbul University-Cerrahpasa; Department of Neuroscience, Institute of Neurological Sciences,

Rapid and Effective Vitamin D Supplementation May Present Better Clinical Outcomes in COVID-19 (SARS-CoV-2) Patients by Altering Serum INOS1, IL1B, IFNg, Cathelicidin-LL37, and ICAM1

Rapid and Effective Vitamin D Supplementation May Present Better Clinical Outcomes in COVID-19 (SARS-CoV-2) Patients by Altering Serum INOS1, IL1B, IFNg, Cathelicidin-LL37, and ICAM1

Background: We aimed to establish an acute treatment protocol to increase serum vitamin D, evaluate the effectiveness of vitamin D3 supplementation, and reveal…

www.mdpi.com

Time for a COVID-19 Vaccine Adverse Event Patient Advocacy National or Even International Group

Time for a COVID-19 Vaccine Adverse Event Patient Advocacy National or International Group

Recently, hundreds of people marched down the streets of Parma, Italy in an organized event termed online as the “March of the Vaccine Dead.” The

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Recently, hundreds of people marched down the streets of Parma, Italy in an organized event termed online as the “March of the Vaccine Dead.” The attendees walked in a line through the streets—those family members and friends of people holding pictures of loved ones who died, purportedly the victims of adverse events associated with the COVID-19 vaccines. There is no doubt that a growing number of people who have been fully vaccinated have experienced adverse events and even death. These people have been shunned and even branded as anti-vaxxers, even as “traitors” to the collective healthcare of a nation. This type of talk is incredibly destructive and must stop. A patient advocacy group must be formed to represent, lobby, and stand for the millions of people that have been injured or to aid family members of the deceased.

The biggest health crisis in modern history, the response to COVID-19, represents an unprecedented clinical investigation capturing the entire world. Early on, a confluence of factors and forces led leading academic research, apex government research institutes, and the pharmaceutical industry to opt for an unprecedented, unrivaled initiative to develop vaccines that would stop transmission, reduce risk, and lead to herd immunity. Tens if not actually hundreds of billions (including indirect costs) have been allocated by governments to pharmaceutical companies to produce vaccine products that were touted as sort of cures to COVID-19. From the U.S. and Trump’s Operation Warp Speed to Russia and China, big money drove a sort of vaccine-arms race to beat the pathogen.

TrialSite, a pro-business, pro-pharma, pro-vaccine, and importantly, non-partisan media platform also commits to its readers to cover and report on medical research in a transparent and objective manner. Independent, TrialSite persists thanks to reader support. This media isn’t influenced by industry advertisers, for example. With a bias favoring freedom, democracy, and fundamental rights covered in the U.S. Constitution (and other such charters around the world), the seemingly orchestrated, controlled and most certainly biased response to COVID-19 has alarmed many, including TrialSite.

The current situation is complex, involving many moving parts, and continues to unfold, which ultimately necessitates non-partisan human mobilization to confront and pivot from the current direction. A new paradigm is required to take on COVID-19. The current approach isn’t working that well. This doesn’t mean the government-sponsored research doesn’t have an important place moving forward—it does, and pharmaceutical companies must be considered partners, not adversaries.

Rising accounts of adverse events, from blood clots and cardiovascular problems, are too severe to ignore anymore. For nearly the entire year TrialSite has received criticism for covering negative aspects of the vaccines but early on, mounting data points raised red flags. With no industry liability—at least, in America due to the PREP Act, the government under the current POTUS mandates doubled down on a truly questionable and potentially unconstitutional approach to dealing with the virus. For example, using OSHA to order a mandate for companies with 100 employees or more to enforce vaccinations, as well as other edicts involving those instructions that receive Medicare. Of course, the lawsuits are now challenging these orders, with some courts already putting the programs on hold. But what about the vaccine victims? While it’s incredibly politically incorrect now to even utter a connection, the reality is that there are growing numbers of cases of adverse events and mortality.

It’s Time to Help Patients

A nascent movement evolves and unfolds due to several parallel crises at this point in history. Resistance and movement for change will take a myriad of forms, including a “medical freedom” movement, as discussed in TrialSite. But, partisan forces seek to intervene and commandeer these.

TrialSite calls for a COVID-19 vaccine-injured patient advocacy association—a non-profit that needs to be set up and funded to advocate for both patients who have been injured due to vaccination or the family members of the deceased.

While many adverse events and deaths haven’t been proven to be directly caused by the vaccines, enough real-world data indicates the time is now to act. This rapidly growing cohort worldwide includes marginalized victims that did nothing to deserve what is now happening to them. Shamed and devalued, these patients are scorned and attacked for being “anti-vaxxers” or “conspiracy theorists” by an increasingly hostile and angry dominant culture that has been conditioned to think that COVID-19 persists because everyone isn’t vaccinated.

But, with mounting breakthrough infections, not to mention continuously mutating variants combined with the universal use of what amounts to early-stage, version 1.0 vaccine products, a ticking time bomb means people must organize to ensure that the patients and family members of the deceased are represented in society.

Answers to complex problems aren’t always simple or easy. In the current unfolding dynamic –and dangerous—situation, people must drop partisan hang-ups and come together to support those who have been injured or family members who have died.

In parallel, a lot has been learned about COVID-19 over the past eighteen months. With rich data indicating early treatment combined with powerful monoclonal antibodies, antivirals, and promising off-label treatments such as Fluvoxamine, in addition to the smart, tailored, and importantly, risk-based use of the COVID-19 vaccines, the NIH and medical establishment must pivot from the existing trajectory. For example, before someone gets their booster, they should have an antibody test. It could very well be that boosters could hurt some people—the risk-benefit analysis must be better understood by all.

TrialSite suspects a seasonal COVID-19 vaccine shot will become the norm much like the influenza vaccine as the products mature and stabilize. A lot of innovation and potential for therapy breakthroughs will undoubtedly result from the COVID-19 pandemic—and that’s a very good thing. A dynamic and thriving biopharma sector benefits societies in countless ways.

But the human cost of this current breakthrough innovation must be accounted for. Therefore, a vaccine adverse event patient advocacy organization needs to be established to bring a collective voice, empowerment, and dignity to what will amount over the next years to tens of millions of people in America alone.

Quote from JedRothwell

Perhaps people in Bangladesh are often infected with parasites, but not diagnosed. I wouldn't know about that. Perhaps the population there is young, so not many people die. Or, perhaps it is like Uttar Pradesh in India insofar as the statistics are meaningless, and deaths are not even detected, or they are covered up.

I don't know why the numbers are low, but I am sure that ivermectin has no effect on COVID. That is what the double-blind tests show. You cannot argue with that. If there was a significant effect, it would be measurable.

Ok how about Mexico city

Ivermectin and the odds of hospitalization due to COVID-19: evidence from a quasi-experimental analysis based on a public intervention in Mexico City

Ivermectin and the odds of hospitalization due to COVID-19: evidence from a quasi-experimental analysis based on a public intervention in Mexico City

Objective To measure the effect of Mexico City’s population-level intervention –an ivermectin-based Medical Kit – – in hospitalizations during the COVID-19…

osf.io

Abstract

Objective

To measure the effect of Mexico City’s population-level intervention –an ivermectin-based Medical Kit – – in hospitalizations during the COVID-19 pandemic.

Methods

A quasi-experimental research design with a Coarsened Exact Matching method using administrative data from hospitals and phone-call monitoring. We estimated logistic-regression models with matched observations adjusting by age, sex, COVID severity, and comorbidities. For robustness checks separated the effect of the kit from phone medical monitoring; changed the comparison period; and subsetted the sample by hospitalization occupancy,

Results

We found a significant reduction in hospitalizations among patients who received the ivermectin-based medical kit; the range of the effect is 52%- 76% depending on model specification.

Conclusions

The study supports ivermectin-based interventions to assuage the effects of the COVID-19 pandemic on the health system.

Quote from Fm1

Oh please provide the one study that shows no effect and I'll show you the now 67 studies that do show a large effect.

I believe the 29 biggest studies that ivermectin enthusiasts (https://ivmmeta.com/) recommend are analyzed here:

Ivermectin: Much More Than You Wanted To Know

...

astralcodexten.substack.com

As you see, these studies are invalid.

Quote from Wyttenbach

https://assets.publishing.serv…llance-report-week-48.pdf

New 48 week report UK.

The boosters show some effect in the age group 70+/80+ with less case cases in ICU but no effect on death rate - why?!

The report also claims Moderna is at least 4x better than Pfizer that is better that Oxford Astra crap.

Further there is a clear signal for pre-time birth among vaccinated woman and in correlation ultra low birth weight. Here I would like to see the absolute numbers.

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That is because you consistently like looking at random raw data and drawing wrong conclusions. No idea why when the various statistical fallacies have been explained to you very well. You have never critiqued or objected to these explanations. It is as though you are incapable of understanding statistics: and incapable of realising that lack. Weird.

Transparency and data – UKHSA’s vaccines report - UK Health Security Agency

ukhsa.blog.gov.uk

Here is a blog that will interest others on why the UK continues to publish raw data and also why it is often abused by antivaxxers. I applaud their stance.

THH

S.Africa's health body sees threefold higher risk of reinfection from Omicron | Reuters

JOHANNESBURG, Dec 2 (Reuters) - The new Omicron variant of the coronavirus poses a threefold higher risk of reinfection than the currently dominant Delta variant and the Beta strain, a group of South African health bodies said on Thursday.

The South African Centre for Epidemiological Modelling and Analysis (SACEMA) and the National Institute of Communicable Diseases (NICD) said the latest findings "provide epidemiological evidence for Omicron's ability to evade immunity from prior infection".

Increased risk of SARS-CoV-2 reinfection associated with emergence of the Omicron variant in South Africa