Its a great shame that bullshitting isn’t an olympic sport, as Switzerland would likely have tripled their paltry medal tally on the last couple of days action here.

Here’s the latest round up of Wyttenfacts, in case you didn’t manage to spot them yourselves…

Quote from Wyttenbach

Only the state of Kerala is missing all targets

Um what?! See Arunachal Pradesh, Manipur, Meghalaya, Mizoram, Sikkim, Tripura… All of which are handing out ivermectin to all and sundry.

Quote from Wyttenbach

and the south (Kerala) is a poor state

Kerala is the 9th richest state out of 33, (GDSP per capita), and the average Keralite is four times wealthier than someone from UP. Also, the south of India is generally richer than the north.

Quote from Wyttenbach

See Uttar Pradesh, the best example! 1000x better than vaccines. But your buddies don't like real data as this cannot be faked as usually done in an RCT study.

UP only registers 60% of deaths in a good year, never mind during a pandemic! Holding them up as providing “real data” shows that someone is well out of their depth.

Or any statistics from the rest of India, for that matter:

Explained: How Indian states misreport their Covid-19 death toll

A new analysis says the actual number of Covid-19 deaths by May 5 could have been as high as 6.54 lakh, not the official tally of 2.21 lakh.

scroll.in

Quote from Wyttenbach

Total Swiss RNA gen therapy deaths so far: 128

Let’s have a look at the link provided, shall we?

“In 128 serious cases, the people concerned died at differing intervals after receiving the vaccine. Despite a chronological correlation, there is no concrete evidence to suggest that the vaccination was the cause of death.”

And you wonder why he doesn’t like posting links… Once more, hanged with his own rope. Wyttenfact status confirmed!

Quote from Wyttenbach

[Wytten]Fact is Delta is much more a cold than a serious infection.

These arguments seem to be based on conventional wisdom and a large measure of hope. (Similar to the apparently-over-optimistic hope that ivermectin is going to save your old anti-vaxxer ass…)

Fortunately we’ve moved beyond 19th-century theorising, and can now instantly read the latest science on the subject:

1.  SARS-CoV-2 Delta VOC in Scotland: demog

2.  How the coronavirus infects cells — and why Delta is so dangerous

3.https://www.bloomberg.com/news…variant-being-more-severe

Might want to try it sometime, eh Wyttenbach?

There’s one or two more Wyttenfacts, but I’m losing patience, and frankly I can’t be bothered explaining why mRNA vaccines aren’t a gene therapy, to a person who claims he can cure cancer with a magic plant, or thinks that inhaling ozone is a good idea - as he is clearly too far around the bend to understand even the basics of medical science.

ANTI-MASKER SENT TO MENTAL HOSPITAL

After an arrest for being on a train without a wearing a mask, he was bailed and invited to explain to the local court why he wasn’t a public nuisance. He turned up to court, again without a mask, so had his bail revoked. After chilling in the cooler for a fortnight his next court appearance was pretty wild…

Man seen without mask on train remanded at IMH after rant during first day of trial

SINGAPORE: A man who was seen without a mask on a train was ordered by a judge on Thursday (Aug 5) to be remanded at the Institute of Mental Health (IMH) after…

www.channelnewsasia.com

It is maybe not surprising that even people who do not go to hospital with COVID can have some long-term cognitive deficit. This study is using objectives measures of cognitive performance - they give similar results to the self-reported effects.

COVID - remember - causes a whole load of things that could affect the brain function:

• general inflammation
• hypoxia
• some evidence of specific attack on nerons (anosmia etc, and various people proposing possible mechanisms)

Put this on the serious unknown side of the do I want me or my family to catch COVID, or be vaccinated, balance. This is not saying everyone has such deficits, but the effect is significant as an overall average - which means it is common:

We know the effect is real, and long term. At the moment there is some evidence of a slow recovery trajectory (much as with strokes). This will be variable. And the mechanism, or mechanisms, are not well understood.

Some people might say that anti-vaxxers suffer cognitive underperformance anyway - so a bit more will not matter. I would not go that direction, and anyway most of the people on the edge of hesiatncy influenced by anti-vas propaganda are juts normal people, reating as anyone would to diverse sources of information when they are not sure who to trust.

DEFINE_ME

Cognitive deficits in people who have recovered from COVID-19

Background

There is growing concern about possible cognitive consequences of COVID-19, with reports of ‘Long COVID’ symptoms persisting into the chronic phase and case studies revealing neurological problems in severely affected patients. However, there is little information regarding the nature and broader prevalence of cognitive problems post-infection or across the full spread of disease severity.

Methods

We sought to confirm whether there was an association between cross-sectional cognitive performance data from 81,337 participants who between January and December 2020 undertook a clinically validated web-optimized assessment as part of the Great British Intelligence Test, and questionnaire items capturing self-report of suspected and confirmed COVID-19 infection and respiratory symptoms.

• View related content for this article

Findings

People who had recovered from COVID-19, including those no longer reporting symptoms, exhibited significant cognitive deficits versus controls when controlling for age, gender, education level, income, racial-ethnic group, pre-existing medical disorders, tiredness, depression and anxiety. The deficits were of substantial effect size for people who had been hospitalised (N = 192), but also for non-hospitalised cases who had biological confirmation of COVID-19 infection (N = 326). Analysing markers of premorbid intelligence did not support these differences being present prior to infection. Finer grained analysis of performance across sub-tests supported the hypothesis that COVID-19 has a multi-domain impact on human cognition.

Interpretation

Interpretation. These results accord with reports of ‘Long Covid’ cognitive symptoms that persist into the early-chronic phase. They should act as a clarion call for further research with longitudinal and neuroimaging cohorts to plot recovery trajectories and identify the biological basis of cognitive deficits in SARS-COV-2 survivors.

Funding

Funding. AH is supported by the UK Dementia Research Institute Care Research and Technology Centre and Biomedical Research Centre at Imperial College London. WT is supported by the EPSRC Centre for Doctoral Training in Neurotechnology. SRC is funded by a Wellcome Trust Clinical Fellowship 110,049/Z/15/Z. JMB is supported by Medical Research Council (MR/N013700/1). MAM, SCRW and PJH are, in part, supported by the National Institute for Health Research (NIHR) Biomedical Research Centre at South London and Maudsley NHS Foundation Trust and King's College London

Quote from Alan Smith

Ring-a-ring-a-roses

A pocket full of posies

Atishoo, atishoo

We all fall down

Posies were thought to have medicinal qualities.

In the U.S. that's "ashes, ashes, we all fall down."

I have read this is not actually an ancient nursery rhyme. It seems to reflect the medieval history of plagues, but modern folklorists say it does not. See:

Ring Around the Rosie: Metafolklore, Rhyme and Reason | Folklife Today

It might as well reflect these things. If it did not originally, it does now.

A little experiment. Enter the following into a google search bar :

german newspaper editor apologizes to children over covid restrictions

and see what you get.

Then go to duckduckgo.com and enter the same thing.

If your results were like mine, you observed that the google input resulted in a low energy, high noise output, while the same duckduckgo input resulted in a much higher energy, coherent output.

Results not like yours. Google has two coherent low quality links:

In Germany, vaccine fears spark conspiracy theories | DW | 12.05.2020

Increasing numbers of Germans are turning out to protest the country's coronavirus measures. Among them are many with a strong suspicion of vaccines — even…

www.dw.com

Why are these top-two google links of low journalistic quality?

greatgameindia.com greatexpose.co.uk

duckduckgo does NOT have them:

Following the greatgameindia link we get, at the bottom:

We need your support to carry on our independent and investigative research based journalism on the Deep State threats facing humanity. Your contribution however small helps us keep afloat. Kindly consider supporting GreatGameIndia.

It clearly has an agenda! This is a pattern, anti-vax, anti-lockdown, anti-science messenging in which the severity of COVID is underestimated, and the dangers of vaccines overestimated, because of an overarching distrust of a deep state.

There are serious and difficult issues here to debate; what is the balance between children having a more normal environment, and a 2% or so avoidable death rate amongst grandparents. It is a very tough situation.

Luckily, with the vaccine rollout, we have a way out of this.

FLCCC weekly update

Quote from THHuxleynew

Results not like yours. Google has two coherent low quality links:

Interesting. My google had nothing of the sort.

My Duckduckgo was different as well. It had these two links showing up first :

German Newspaper's Editor Apologizes for Spreading COVID-19 Propaganda | InfoArmed

Julian Reichelt, the editor-in-chief of the popular German newspaper Bild—with a current circulation of about 1.37 million copies daily—has made a public…

infoarmed.com

Editor-In-Chief Of Germany’s Top Newspaper Apologizes For Fear-Driven COVID Coverage by Paul Joseph Watson – EarthNewspaper.com – All The Honest News Fit To Publish

Here's a youtube video link from the first article.

MarkU…

I get similar results to Huxley but the order is slightly different. DDG has same results for UK vs. All regions

Quote from Alan Smith

The results included the failed First Anglo-Afghan War of 1838, the First Anglo-Sikh War of 1845, the Second Anglo-Sikh War of 1848, the Second Anglo-Afghan War of 1878, and the annexation of Kokand by Russia.

What was the final score? Who won?

Enjoy

Bild, Merkel and the culture wars: the inside story of Germany’s biggest tabloid

Context on Mark U's "Oh my god - the deep state is hiding something" link.

Reichelt, who is 40, made his name as a war reporter in Syria, but today confines most of his battle courage to Twitter, where he enjoys needling the German political establishment and barging into leftwing echo chambers. In person, Reichelt exudes a twitchy exuberance, like a fighter pilot who has managed to smuggle champagne into the cockpit. His eyes restlessly gauge the world around him, clocking who he needs to avoid and who he needs to attract. Into his phone, he volleys directives to subeditors, assistants and the band of young male disciples he sends around the world to collect stories. “The leading populist in western Germany,” is how Albrecht von Lucke, editor of the prestigious left-liberal monthly Blätter, describes him.

Despite its traditional proximity to power, Bild has mutated into something beyond a stalwart defender of the German status quo. Now, says Joffe, it is “an equal-opportunity anti-establishment force”. “Previous Bild editors made their alliances more based on personal feeling,” says the journalist Stefan Niggemeier, who in 2004 co-founded the BILDBlog, a website that corrects Bild’s factual errors and misrepresentations. “But Reichelt is different. He is an actually political person.”

Reichelt’s agenda is marked less by novelty than by a chest-crunching resuscitation of Bild’s core commitments: pro-US, pro-Nato, pro-Israel, pro-austerity, pro-capital, anti-Russia, anti-China. According to the Bild worldview, the best way to counter the left is to portray its demands as totalitarian, and the best way to kill off the far right is to cannibalise its grievances. While Bild prints relatively little material that a supporter of the far-right Alternative for Germany (AfD) party would object to, Reichelt sees the party as a threat to his effort to remake the German political scene. “We want nothing to do with the imbeciles of the AfD,” he told me. “The way to destroy them is to make room for their voters in what used to be the political mainstream of this country.”

Bild’s main attack targets remain Germany’s “Gutmenschen” – the do-gooders, vegetarians, Greens and 1968ers who are treated as parasites and irritants to Germany’s robust economy and middle class. More recently, the paper’s sights have also turned on Merkel and her party, the Christian Democratic Union (CDU). “The paper’s constant targeting of Merkel, but also its current take on refugees and all sorts of minorities definitely reproduces rhetorical elements of the AfD,” says Fatma Aydemir, a novelist and columnist for the leftwing daily Tageszeitung, known as Taz. In the absence of serious challenges from within Merkel’s own party, or from other parties – she has been chancellor for 14 years, a tenure surpassed only by Kohl and Bismarck – Bild has attempted to make itself into that opposition. Yet any populist challenger finds itself in the hard position of convincing Germans that Merkel’s very successful form of reactive politics has run out of historical steam. “We are the voice of ordinary people,” claims Reichelt. “If we didn’t exist, they would really think the whole system is against them.”

On the environment, one day Bild might be lamenting Merkel’s call to shutter coal factories by 2038 (“The coal deadline is going to cost us!” ran a headline about the prospective job losses.) But another day you might read that “Our government officials are destroying the environment!” atop an article detailing the 200,000 yearly flights German officials take between ministries in Berlin and Bonn. Like Merkel herself, Reichelt is an arch-opportunist, congenitally averse to letting crises go to waste.

As editor, Reichelt sees himself less as a news impresario than as an emotional entrepreneur. “Journalism is basically about emotions, as all of the other news outlets in this country seem to have forgotten,” he told me. Reichelt likes to point out what he sees as the shared delusions of the more “respectable” German press. He gave the example of Merkel, around whom he said the press had created an “elaborate mythology” that she has such natural wit and is extremely clever, whereas her skill lay in identifying the direction of the prevailing winds.

Quote from Alan Smith

As some of you may know, 'The Great Game' was an agenda all by itself. Wiki says.

"The Great Game" was a political and diplomatic confrontation that existed for most of the 19th century and beginning of the 20th century between the British Empire and the Russian Empire, over Afghanistan and neighbouring territories in Central and South Asia. It also had direct consequences in Persia and British India. Britain was fearful that Russia planned to invade India and that was the goal of its expansion in central Asia. As a result, there was a deep atmosphere of distrust and the talk of war between the two major European empires.[1][2][3] Britain made it a high priority to protect all the approaches to India, and the "great game" is primarily how the British did this. Most historians of Russia have concluded that after 1801 Russia never had any intention or plan involving India, as the Russians repeatedly stated to the highly suspicious British.[4]

The Great Game began on 12 January 1830 when Lord Ellenborough, the President of the Board of Control for India, tasked Lord William Bentinck, the Governor-General, with establishing a new trade route to the Emirate of Bukhara.[2][3][5] Britain intended to gain control over the Emirate of Afghanistan and make it a protectorate, and to use the Ottoman Empire, the Persian Empire, the Khanate of Khiva, and the Emirate of Bukhara as buffer states blocking Russian expansion. This would protect India and also key British sea trade routes by stopping Russia from gaining a port on the Persian Gulf or the Indian Ocean.[2][3] Russia proposed Afghanistan as the neutral zone.[6] The results included the failed First Anglo-Afghan War of 1838, the First Anglo-Sikh War of 1845, the Second Anglo-Sikh War of 1848, the Second Anglo-Afghan War of 1878, and the annexation of Kokand by Russia.

If you've read Kim you know that of course...

Birds Dying and no known cause.

No Evidence of Mysterious Bird Disease in Mass., Officials Say

Bird feeders have grown in popularity in lockstep with the emergence of work-from-home schedules for many people in Massachusetts, but state officials are…

www.nbcboston.com

Structural basis for enhanced infectivity and immune evasion of SARS-CoV-2 variants

Structural basis for enhanced infectivity and immune evasion of SARS-CoV-2 variants

As battles to contain the COVID-19 pandemic continue, attention is focused on emerging variants of the severe acute respiratory syndrome coronavirus 2…

science.sciencemag.org

SARS-CoV-2 from alpha to epsilon

As battles to contain the COVID-19 pandemic continue, attention is focused on emerging variants of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) virus that have been deemed variants of concern because they are resistant to antibodies elicited by infection or vaccination or they increase transmissibility or disease severity. Three papers used functional and structural studies to explore how mutations in the viral spike protein affect its ability to infect host cells and to evade host immunity. Gobeil et al. looked at a variant spike protein involved in transmission between minks and humans, as well as the B1.1.7 (alpha), B.1.351 (beta), and P1 (gamma) spike variants; Cai et al. focused on the alpha and beta variants; and McCallum et al. discuss the properties of the spike protein from the B1.1.427/B.1.429 (epsilon) variant. Together, these papers show a balance among mutations that enhance stability, those that increase binding to the human receptor ACE2, and those that confer resistance to neutralizing antibodies

Abstract

Several fast-spreading variants of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) have become the dominant circulating strains in the COVID-19 pandemic. We report here cryo–electron microscopy structures of the full-length spike (S) trimers of the B.1.1.7 and B.1.351 variants, as well as their biochemical and antigenic properties. Amino acid substitutions in the B.1.1.7 protein increase both the accessibility of its receptor binding domain and the binding affinity for receptor angiotensin-converting enzyme 2 (ACE2). The enhanced receptor engagement may account for the increased transmissibility. The B.1.351 variant has evolved to reshape antigenic surfaces of the major neutralizing sites on the S protein, making it resistant to some potent neutralizing antibodies. These findings provide structural details on how SARS-CoV-2 has evolved to enhance viral fitness and immune evasion.

Discussion

Transmissibility and immune evasion are independent selective forces driving emergence of viral genetic diversity. The changes of most concern in the SARS-CoV-2 S protein would be those that simultaneously enhance transmission, augment disease severity, and evade immune recognition in previously exposed hosts. Our data suggest that the most problematic combination of such mutations is not yet present in the existing variants examined here.

In the B.1.1.7 virus, mutations A570D and S982A lead to an outward shift of the CTD1, thereby relaxing the FPPR and 630 loop, which help retain the RBD in its “down” position in the parental strain. The mutations increase the frequency with which the S trimer samples the RBD-up conformation, allowing B.1.1.7 to better present the receptor binding motif (RBM) to ACE2 on the host cells. Once one RBD flips up, the fully or partially ordered 630 loops of the neighboring protomers stabilize the CTD2, which folds together with the N-terminal segment of S2, and thus prevent the premature S1 dissociation. N501Y in the ACE2 binding site of the RBD also increases the affinity of that domain for the receptor, probably because of the hydrophobic interaction of Tyr501 with Tyr41 of ACE2 (36) and a possible cation-π interaction with ACE2 Lys353 (fig. S16). The combination of enhanced RBM presentation and additional local interactions might allow the B.1.1.7 virus to infect cell types with lower ACE2 levels than those of the nasal and bronchial epithelial cells that the virus typically infects; an expanded cell tropism could account for the increased risk of mortality in patients infected with this variant (9, 10). The mutations in B.1.1.7 caused no major structural rearrangements in the RBD and NTD, consistent with minimal changes in the sensitivity of the B.1.1.7 variant to the potently neutralizing antibodies [tables S1 and S2; (33)].

In the B.1.351 virus, the S protein largely retains the structure of the G614 trimer with almost identical biochemical stability. N501Y, K417N, and E484K in the RBD have not caused major structural changes, but the loss of salt bridges between Lys417 and ACE2 Asp30 and Glu484 and ACE2 Lys31 mitigates the increased receptor affinity imparted by N501Y (fig. S16). K417N and E484K probably lead to loss of binding and neutralization by antibodies that target the RBD-2 epitopes (fig. S4A). The accompanying mutations in the NTD remodel the antigenic surface and greatly reduce the potency of neutralizing antibodies against NTD-1 epitopes. The B.1.351 variant was probably selected under a certain level of immune pressure, because it altered two major neutralizing sites on the S trimer simultaneously with only a slight compromise in its ability to engage a host cell.

The global range of SARS-CoV-2 and the daily vast number of replication events make emergence of new variants inevitable and substantially increases the viral genetic diversity. In many cases, antibody resistance may compromise viral fitness, as in the B.1.351 variant, which resists neutralization by RBD-directed antibodies but also loses the enhanced affinity and transmissibility imparted by N501Y, as a consequence of the immune-escape mutations. It is also possible to combine immune evasion and virulence through continuous viral evolution, such as a B.1.1.7 variant that contains the E484K mutation (B.1.1.7+E484K) (41). Such a combination will bring greater challenges for vaccine development compared with the beginning of the pandemic. If SARS-CoV-2 becomes seasonal, innovative strategies already developed against other human pathogens—such HIV-1, hepatitis C virus, and influenza virus—may be applicable to on-going control of the COVID-19 pandemic. The B.1.351 S trimer, which has superior biochemical stability and new epitopes, should be an excellent starting point for developing next-generation vaccines designed to elicit broadly neutralizing antibody responses

Quote from sam12

Birds Dying and no known cause.

https://www.nbcboston.com/news…ss-officials-say/2453627/

perhaps it is mRNA vaccines altering their genes?

86% of Youth Getting First Pfizer COVID-19 Shot Experienced Adverse Reactions According to FDA Data

86% of Youth Getting First Pfizer COVID-19 Shot Experienced Adverse Reactions According to FDA Data

According to The Daily Expose, a “Shocking 86% of Children suffered an Adverse Reaction to the Pfizer Covid Vaccine in Clinical Trial.” Some consider this

trialsitenews.com

According to The Daily Expose, a “Shocking 86% of Children suffered an Adverse Reaction to the Pfizer Covid Vaccine in Clinical Trial.” Some consider this particular outlet to be fringe, so we can compare their version of the facts with the June FDA EUA Fact Sheet’s description of the data which the article relied on. On May 10, the FDA granted a EUA for Pfizer’s mRNA COVID-19 vaccine for folks aged 12 and up. Pfizer’s clinical trials on 12-15 years old showed that 86% of subjects had an adverse response from mild to serious. This data can be found in the FDA Fact Sheet on page 25, Table 5 onwards. These tables show that 1,127 kids got the first shot and 1,097 got the second dose. The Daily Expose implies that the missing 30 kids had adverse effects, but this claim is not sourced. 86% of those getting the first shot had an adverse reaction. 78.9% of those getting the second shot responded similarly.

According to Table 6 in the Fact Sheet, 20.3% of kids who got the first shot got a fever, while 39.3% of those getting the second shot got the same side effect. 60.1% of kids had fatigue from the first shot; the figure is 66% for the second dose. 55.3% had a headache after the first shot, and 64.5% did so after the second shot. Other problems were chills, 27.6% for the first dose and 41.5% for the second; vomiting, 2.8% for the first dose and 2.6% for the second; and diarrhea, 8.0% for the first shot and 5.9% for the second shot. Per FDA, 0.04% of kids had an “extremely serious adverse reaction” of an unspecified nature. If all four million 12–15 year-olds in the UK were given this shot, we could “expect to see 1,600 suffer an extremely serious adverse reaction which could include death.” The Daily Expose points out that we need to compare these risks to the actual risk to kids from COVID-19. Given that youth don’t tend to get very sick from this pandemic, we should ask why we need to vaccinate them if real risks are involved?

Quote from Fm1

86% of Youth Getting First Pfizer COVID-19 Shot Experienced Adverse Reactions According to FDA Data

https://trialsitenews.com/86-o…ns-according-to-fda-data/

According to The Daily Expose, a “Shocking 86% of Children suffered an Adverse Reaction to the Pfizer Covid Vaccine in Clinical Trial.” Some consider this particular outlet to be fringe, so we can compare their version of the facts with the June FDA EUA Fact Sheet’s description of the data which the article relied on. On May 10, the FDA granted a EUA for Pfizer’s mRNA COVID-19 vaccine for folks aged 12 and up. Pfizer’s clinical trials on 12-15 years old showed that 86% of subjects had an adverse response from mild to serious. This data can be found in the FDA Fact Sheet on page 25, Table 5 onwards. These tables show that 1,127 kids got the first shot and 1,097 got the second dose. The Daily Expose implies that the missing 30 kids had adverse effects, but this claim is not sourced. 86% of those getting the first shot had an adverse reaction. 78.9% of those getting the second shot responded similarly.

According to Table 6 in the Fact Sheet, 20.3% of kids who got the first shot got a fever, while 39.3% of those getting the second shot got the same side effect. 60.1% of kids had fatigue from the first shot; the figure is 66% for the second dose. 55.3% had a headache after the first shot, and 64.5% did so after the second shot. Other problems were chills, 27.6% for the first dose and 41.5% for the second; vomiting, 2.8% for the first dose and 2.6% for the second; and diarrhea, 8.0% for the first shot and 5.9% for the second shot. Per FDA, 0.04% of kids had an “extremely serious adverse reaction” of an unspecified nature. If all four million 12–15 year-olds in the UK were given this shot, we could “expect to see 1,600 suffer an extremely serious adverse reaction which could include death.” The Daily Expose points out that we need to compare these risks to the actual risk to kids from COVID-19. Given that youth don’t tend to get very sick from this pandemic, we should ask why we need to vaccinate them if real risks are involved?

Display More

I don't actually understand the point of this story, except to reveal that populist summaries of medical tests are pretty rubbish.

Is there anything surprising int most children (young immune system) getting transient side effects after a vaccine - at a level slightly worse than adults?

From the fact sheet:

In clinical studies, adverse reactions in participants 16 years of age and older included pain at the injection site
(84.1%), fatigue (62.9%), headache (55.1%), muscle pain (38.3%), chills (31.9%), joint pain (23.6%),
fever (14.2%), injection site swelling (10.5%), injection site redness (9.5%), nausea (1.1%), malaise (0.5%), and
lymphadenopathy (0.3%).

In a clinical study, adverse reactions in adolescents 12 through 15 years of age included pain at the injection site
(90.5%), fatigue (77.5%), headache (75.5%), chills (49.2%), muscle pain (42.2%), fever (24.3%), joint pain
(20.2%), injection site swelling (9.2%), injection site redness (8.6%), lymphadenopathy (0.8%), and nausea
(0.4%).

Quote from Fm1

Given that youth don’t tend to get very sick from this pandemic, we should ask why we need to vaccinate them if real risks are involved?

Answer: These are not real risks. They are expected side-effects. They indicate that the vaccine is working correctly. Many childhood vaccinations cause similar side effects in infants and small children. It would be insane to stop giving them, because the risk of a serious problem from the vaccine is orders of magnitude smaller than the risks from the disease. People who ask questions like this are ignorant of basic science and statistics.

Risks of a vaccine reaction

For DTaP vaccine:

• Soreness or swelling where the shot was given, fever, fussiness, feeling tired, loss of appetite, and vomiting sometimes happen after DTaP vaccination.

This is trying for the parent and child, but nothing to worry about. It is not a "real risk." In rare cases, more dangerous reactions occur:

• More serious reactions, such as seizures, non-stop crying for 3 hours or more, or high fever (over 105°F) after DTaP vaccination happen much less often. Rarely, the vaccine is followed by swelling of the entire arm or leg, especially in older children when they receive their fourth or fifth dose.

Such outcomes have not been reported for COVID vaccines.

As noted above by THH, the side effects in children are only slightly more severe than in adults, which is not surprising.