Covid-19 News

Quote from Wyttenbach

Far more than 1000 deaths from vaccines after 100 mio. doses. In Switzerland alone we had about 180 severe incidents with less than 1 mio. doses. Pfizer vaccines cause CoV-19. Thus we have an unaccounted number of people that dies from vaccine induced CoV-19. Some 100 in Israel alone.

Does a Pfizer vaccination really ends up with an infection due to the vaccine itself (excluding any other contact with external Corona virus!), or is it just blocking or reducing the immune response shortly after the jab so people can catch the SRASS-CoV2 easier?

Is there any thinkeable biological process, that this mRAN vaccine induces the cells to creates real Corona virus inside your body? That should be evaluated. If so vaccinated people can become superspreaders, couldn't they?

Interesting read though, despite its big pharma mafia origin...

https://www.pfizer.com/news/pr…high-effectiveness-pfizer

Quote from zorud

Does a Pfizer vaccination really ends up with an infection due to the vaccine itself (excluding any other contact with external Corona virus!),

No way.

Quote from zorud

or is it just blocking or reducing the immune response shortly after the jab so people can catch the SRASS-CoV2 easier?

Very possible. I'm listening to a radio talk show host right now (Jerry Agar) whose healthy adult daughter got very ill for three days after her second moderna vaccine. Then it cleared, but then she got even sicker again, for longer. Went to the hospital to get checked for strep throat and it turns out she had Covid. Moral of the story: any vaccine or pathogen that gets you sick leaves you vulnerable to a new illness.

Quote from zorud

Is there any thinkeable biological process, that this mRAN vaccine induces the cells to creates real Corona virus inside your body?

No way. Only the spike protein. Problem is, the spike protein by itself, without the live virus, has been implicated in brain inflammation and blood clotting.

Quote from zorud

Interesting read though, despite its big pharma mafia origin...

https://www.pfizer.com/news/pr…high-effectiveness-pfizer

A rule for vaccination is that the person getting vaccinated should not be currently fighting an infection. (Dirty little secret : our body is often fighting something, unbeknownst to the doctor!) So, people who are suspected of having Covid or another infection will (should) not be getting vaccinated. This produces what is called a 'health bias', because the unvaccinated sick are more likely to get Covid than the healthy vaccinated, thus misleadingly inflating the vaccine efficacy number.

Digging into uk study of B117

Quote from zorud

Does a Pfizer vaccination really ends up with an infection due to the vaccine itself (excluding any other contact with external Corona virus!), or is it just blocking or reducing the immune response shortly after the jab so people can catch the SRASS-CoV2 easier?

The vaccine makes the immune system significantly more tolerant for the CoV-19 virus during the first 10 days (Day 7 peek). How in details this effect works must be more deeply investigated. I think (guess) the immune system believes it is already reacting and delays the production of antibodies or the T-Cell act the wrong way as long as the vaccine is working.

Pfizer cheated these jab induced CoV-19 cases (>200) away in the phase III study. (All documented and has been linked here)

Do not believe any data Pfizer publishes. This company together with Gilead is among the most corrupt on the market. All they say is just marketing- trying to boost sales. And they also are an acting part of the current world wide mass murder by bribing people for not allowing Ivermectin.

True data will only be available once the lock down ends and 3 more months have passed.

Quote from zorud

Does a Pfizer vaccination really ends up with an infection due to the vaccine itself (excluding any other contact with external Corona virus!),

No, that is physically impossible. Next question.

Long Covid isn’t as unique as we thought

The nagging symptoms long-haulers experience reveal a frustrating blind spot in medicine.

https://www.vox.com/22298751/l…-covid-19-hauler-symptoms

Dr. Craig Spencer, an emergency room doctor in New York City, was no stranger to dangerous viruses when a brush with one landed him in Bellevue Hospital for 19 days. But it was only after he was discharged, and declared virus-free, that the really bizarre symptoms set in.

Back at home, he noticed he couldn’t taste anything for several days. For months, he was tired all the time and his joints felt heavy and painful. When he woke up in the morning, his back was “stiff like a bamboo rod.” His weight dropped, and clumps of hair fell out. Though the physical effects eventually faded, cognitive complications persist to this day — what he describes as “a subtle but noticeable difference in concentration and ability to form new memories.”

If Spencer’s constellation of ongoing symptoms — fatigue, muscle and joint pain, memory issues — sounds familiar, it’s because it has become a frightening feature of some coronavirus infections, an epidemic of long-term illness within the pandemic. For the Covid-19 “long-haulers,” symptoms can persist for weeks or even months, long after being discharged from the hospital or testing positive for the virus, if they even saw a doctor or got diagnosed at all.

But Spencer never had Covid-19. His persistent aches, pains, and memory problems arose after contracting Ebola in late 2014, when he was working with Doctors Without Borders in Guéckédou, Guinea, the epicenter of the West Africa Ebola epidemic. The experience has led him to join the growing chorus of health professionals, patient advocates, and researchers who argue we need to reframe how we think about coronavirus long-haulers.

The dominant narrative about long Covid has been that it’s a uniquely perplexing feature of Covid-19. Reports of “Covid brain fog” or “Covid dementia,” for example, suggest a disturbing and extraordinary ability of the coronavirus to destroy the lives of survivors. Even a year later, some patients are still struggling to return to work or have their illness recognized, let alone access disability benefits.

While there’s no doubt long Covid is a real condition worthy of diagnosis and treatment, “this isn’t unique to Covid,” Akiko Iwasaki, an immunologist at the Yale School of Medicine, said. Rather, Covid-19 appears to be one of many infections, from Ebola to strep throat, that can give rise to stubbornly persistent symptoms in an unlucky subset of patients. “If Covid didn’t cause chronic symptoms to occur in some people,” PolyBio Research Foundation microbiologist Amy Proal told Vox, “it would be the only virus that didn’t do that.”

Even with growing awareness about long Covid, patients with chronic “medically unexplained” symptoms — that don’t correspond to problematic blood tests or imaging — are still too often minimized and dismissed by health professionals. It’s a frustrating blind spot in health care, but one that can’t be as easily ignored with so many new patients entering this category, said Megan Hosey, assistant professor at the Johns Hopkins Department of Physical Medicine and Rehabilitation.

“It has always been [and] is the case that patients who get sick experience high levels of symptoms like those described by long-Covid patients,” she said. “We have just done a terrible job of acknowledging [and] treating them.”

This is called viral persistence. Africa is now seeing this with ebola.

Quote from Fm1

“It has always been [and] is the case that patients who get sick experience high levels of symptoms like those described by long-Covid patients,” she said. “We have just done a terrible job of acknowledging [and] treating them.”

Ivermectin. Every damaged person more will add to the storm the mafia will face soon...

Is the vermectin blend effective for the 117? Seems there is a bit of a info blackout on this..

Quote from DnG

Is the vermectin blend effective for the 117? Seems there is a bit of a info blackout on this..

Ivermectin blocks one crucial RNA strain of the virus from replication and thus is independent of mutations!

To some extent it also work as antibody, what is a problem as a part of the dose goes away "unused". So after a strong infection you must start with at least 2x dose.

The Lopez anti-ivermectin 'in Colombia 'study'

widely publicized.. but shonky .". following the Big Pharma money

meantime the world mediacracy ignores the ivermectin elephant in India ..

https://osf.io/u7ewz/

Given AEs distinctive for IVM use in study controls

and failures of blinding and boundaries between IVM and placebo arms,

results for study controls are unreliable..

Chinese Vacines.

Off topic but interesting video

on the Great Reset.

First Court Case against Mandatory

Vaccination.

Quote from zorud

Interesting read though, despite its big pharma mafia origin...

Some real official inormation from CDC (USA): https://www.cdc.gov/coronaviru…ly-vaccinated-people.html

This is given to relate the Pfizer fake news with the facts.

Real-world vaccine effectiveness

Preliminary analyses from the United States, United Kingdom, and Israel demonstrate that a two-dose mRNA COVID-19 vaccination series is highly effective against SARS-CoV-2 infection (including both symptomatic and asymptomatic infections). In the United States, the effectiveness of mRNA COVID-19 vaccination (either Pfizer-BioNTech or Moderna) was 89% against SARS-CoV-2 infection; vaccinated persons who were diagnosed with COVID-19 had a 60% lower hospitalization rate than unvaccinated persons ¹⁴. Among U.K. healthcare personnel, Pfizer-BioNTech COVID-19 vaccination was 86% effective against SARS-CoV-2 infection ¹⁵. In U.K. adults aged ≥ 80 years, including those with multiple underlying medical conditions, vaccine efficacy against symptomatic disease was estimated at 85% ¹⁶. In Israel, two doses of Pfizer-BioNTech COVID-19 vaccine was 90–94% effective against a spectrum of illness: asymptomatic SARS-CoV-2 infection, symptomatic COVID-19, as well as specifically severe COVID-19 ¹⁷. Preliminary data from Israel suggest that persons vaccinated with Pfizer-BioNTech COVID-19 vaccine who develop COVID-19 have a four-fold lower viral load than unvaccinated persons ¹⁸. This observation may indicate reduced transmissibility, as viral load has been identified as a key driver of transmission ¹⁹.

I have just had the Pfizer/Biontec jab - the mRNA one- had absolutely no effect on me because I was always immune to COVID (all the variants) because I have been taking ANTI-Bat regularly. Governments and drug companies are now glad to declare the pandemic over. They have made their vast profits and have exterminated useless members of their populations. Seig XXXXXX everybody!!! Welcome to the XXXX and World War Three!!

Quote from Toffoli

"Childrens volunteer to test the safety of the covid vaccine"

Children should also take a vaccination against greed, air pollution and fat Hamburgers and free masons...

Another component of anti bat, a welcomed one !!!!!! THE SMOKING LAMP IS LIT!!!

Cannabidiol Inhibits SARS-CoV-2 Replication and Promotes the Host Innate Immune Response

https://www.biorxiv.org/content/10.1101/2021.03.10.432967v1

ABSTRACT

The rapid spread of COVID-19 underscores the need for new treatments. Here we report that cannabidiol (CBD), a compound produced by the cannabis plant, inhibits SARS-CoV-2 infection. CBD and its metabolite, 7-OH-CBD, but not congeneric cannabinoids, potently block SARS-CoV-2 replication in lung epithelial cells. CBD acts after cellular infection, inhibiting viral gene expression and reversing many effects of SARS-CoV-2 on host gene transcription. CBD induces interferon expression and up-regulates its antiviral signaling pathway. A cohort of human patients previously taking CBD had significantly lower SARS-CoV-2 infection incidence of up to an order of magnitude relative to matched pairs or the general population. This study highlights CBD, and its active metabolite, 7-OH-CBD, as potential preventative agents and therapeutic treatments for SARS-CoV-2 at early stages of infection.

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is responsible for coronavirus disease 2019 (COVID-19), a pandemic that has overtaken the world during the past year. SARS-CoV-2, related to severe acute respiratory syndrome-related coronavirus (SARS-CoV), is the seventh species of coronavirus known to infect people. These coronaviruses, which include SARS-CoV, 229E, NL63, OC43, HKU1, and MERS-CoV cause a range of symptoms from the common cold to more severe pathologies (1). Despite recent vaccine availability, SARS-CoV-2 is still spreading rapidly (2), highlighting the need for alternative treatments, especially for populations with limited access to vaccines. To date, few therapies have been identified that block SARS-CoV-2 replication and viral production.

SARS-CoV-2 is a positive-sense single-stranded RNA (+ssRNA) enveloped virus composed of a lipid bilayer and four structural proteins that drive viral particle formation. The spike (S), membrane (M), and envelope (E) are integral proteins of the virus membrane and serve to drive virion budding, while also recruiting the nucleocapsid (N) protein and the viral genomic RNA into nascent virions. Like SARS-CoV, SARS-CoV-2 primarily enters human cells by the binding of the viral S protein to the angiotensin converting enzyme 2 (ACE2) receptor (3–5), after which the S protein undergoes proteolysis by transmembrane protease, serine 2 (TMPRSS2) or other proteases into two non-covalently bound peptides (S1, S2) that facilitate viral entry into the host cell. The N-terminal S1 binds the ACE2 receptor, and the C-terminal S2 mediates viral-cell membrane fusion following proteolytic cleavage by TMRSS2 or other proteases. Depending upon the cell type, viral entry can also occur after ACE2 binding, independent of proteolytic cleavage (6–8). Following cell entry, the SARS-CoV-2 genome is translated into two large polypeptides that are cleaved by two viral proteases, MPro and PLPro (9, 10), to produce 15 proteins, in addition to the synthesis of subgenomic RNAs that encode another 10 accessory proteins plus the 4 structural proteins. These proteins enable viral replication, assembly, and budding. In an effort to suppress infection by the SARS-CoV-2 beta-coronavirus as well as other evolving pathogenic viruses, we tested the antiviral potential of a number of small molecules that target host stress response pathways.

One potential regulator of the host stress and antiviral inflammatory responses is cannabidiol (CBD), a member of the cannabinoid class of natural products (11). CBD is produced by Cannabis sativa (Cannabaceae; marijuana/hemp). Hemp refers to cannabis plants or materials derived thereof that contain 0.3% or less of the psychotropic tetrahydrocannabinol (THC) and typically have relatively high CBD content. By contrast, marijuana refers to C. sativa materials with more than 0.3% THC by dry weight. THC acts through binding to the cannabinoid receptor, and CBD potentiates this interaction (12). Despite numerous studies and many typically unsubstantiated claims related to CBD-containing products, the biology of CBD itself is unclear and specific targets are mostly unknown (11). However, an oral solution of CBD is an FDA-approved drug, largely for the treatment of epilepsy (13). Thus, CBD has drug status, is viable as a therapeutic, and cannot be marketed as a dietary supplement in the United States (11). Although limited, some studies have reported that certain cannabinoids have antiviral effects against hepatitis C virus (HCV) and other viruses (14).

RESULTS

To test the effect of CBD on SARS-CoV-2 replication, we pretreated A549 human lung carcinoma cells expressing exogenous human ACE-2 receptor (A549-ACE2) for 2 hours with 0-10 μM CBD prior to infection with SARS-CoV-2. After 48 hours, we monitored cells for expression of the viral spike protein (S). For comparison, we also treated cells over a similar dose range with an MLK inhibitor (URMC-099) previously implicated as an antiviral for HIV (12) and KPT-9274, a PAK4/NAMPT inhibitor (13) that our analysis suggested might reverse many changes in gene expression caused by SARS-CoV-2. All three inhibitors potently inhibited viral replication under non-toxic conditions with EC50s ranging from 0.2-2.1 μM (Fig. 1A). CBD inhibited SARS-CoV-2 replication in Vero E6 monkey kidney epithelial cells as well (fig. S1A). No toxicity was observed at the effective doses (fig. S1B). We also determined that CBD suppressed replication of a related beta-coronavirus, mouse hepatitis virus (MHV), under non-toxic conditions with an EC50 of ~5 μM using A549 cells that express the MHV receptor (A549-MHVR), indicating the potential for more broader viral efficacy (fig. S1C,D).

Quote from Fm1

Cannabidiol Inhibits SARS-CoV-2 Replication and Promotes the Host Innate Immune Response

Now we understand better why the pharma mafia did declare war on drugs other than authorized by the pharma mafia. ..

Same with the anti cancer potential of methadon and other low cost drugs.